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Sanjay Kaul

Researcher at Cedars-Sinai Medical Center

Publications -  222
Citations -  25862

Sanjay Kaul is an academic researcher from Cedars-Sinai Medical Center. The author has contributed to research in topics: Randomized controlled trial & Myocardial infarction. The author has an hindex of 67, co-authored 215 publications receiving 22556 citations. Previous affiliations of Sanjay Kaul include University of California, Los Angeles & Baylor University Medical Center.

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Current Status and Future Directions in the Controversy Over Stenting

TL;DR: The current status of this controversy is reviewed, the additional evidence needed for its resolution is described, and recommendations for regulatory reform and 3 specific recommendations to encourage evidence-based patient management are offered.
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As Time Goes By: Current Status and Future Directions in the Controversy Over Stenting

TL;DR: In this paper, the authors review the current status of this controversy, describe the additional evidence needed for its resolution, and offer recommendations for regulatory reform and 3 specific recommendations to encourage evidence-based patient management: 1) an emphasis on medical therapies with proven longterm benefit; 2) the use of kinetic modeling to estimate long-term outcomes of therapies based on the available near-term data.
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Comparative Antithrombotic Effects of Magnesium Sulfate and the Platelet Glycoprotein IIb/IIIa Inhibitors Tirofiban and Eptifibatide in a Canine Model of Stent Thrombosis

TL;DR: Magnesium produced a significant reduction in acute stent thrombus formation that was equivalent in magnitude to that produced by clinically relevant doses of tirofiban and eptifibatide, and its potential use in percutaneous coronary intervention requires further study.
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Mitigating Cardiovascular Risk in Type 2 Diabetes With Antidiabetes Drugs: A Review of Principal Cardiovascular Outcome Results of EMPA-REG OUTCOME, LEADER, and SUSTAIN-6 Trials.

TL;DR: The results of these trials yield a favorable benefit-risk balance for these therapies in mitigating CV risk in patients with type 2 diabetes, and more research is needed to elucidate the underlying mechanisms and confirm whether the CV benefits are a class effect or whether the benefits persist in patients without established CV disease or are evident even in patients Without diabetes.