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Sanoara Mazid

Researcher at Cornell University

Publications -  8
Citations -  209

Sanoara Mazid is an academic researcher from Cornell University. The author has contributed to research in topics: Hippocampal formation & Hippocampus. The author has an hindex of 4, co-authored 7 publications receiving 164 citations. Previous affiliations of Sanoara Mazid include Rockefeller University.

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Estrogen receptor alpha and beta specific agonists regulate expression of synaptic proteins in rat hippocampus.

TL;DR: The results support previous reports that estradiol and isoform specific agonists differentially activate ERalpha and ERbeta to regulate protein expression and suggest that the desired therapeutic outcome of estrogen may be accomplished by using specific estrogen receptor agonists.
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Sex differences after chronic stress in the expression of opioid-, stress- and neuroplasticity-related genes in the rat hippocampus.

TL;DR: In this article, a study was conducted to determine if there are also sex differences in gene expression in the hippocampus following chronic immobilization stress (CIS) in a manner that promotes drug-related learning.
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Sex differences in subcellular distribution of delta opioid receptors in the rat hippocampus in response to acute and chronic stress.

TL;DR: Data indicate that DORs redistribute within CA3 pyramidal cells and dentate hilar GABAergic interneurons in a sexually dimorphic manner that would promote activation and drug related learning in males after AIS and in females after CIS.
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Estrogen Receptor β Contributes to Both Hypertension and Hypothalamic Plasticity in a Mouse Model of Peri-Menopause.

TL;DR: In this paper, the role of estrogen receptor β (ERβ) signaling in the hypothalamic paraventricular nucleus (PVN) was investigated in a model of early stage ovarian failure (peri-AOF) characteristic of perimenopause.
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Effects of estrogen and aging on synaptic morphology and distribution of phosphorylated Tyr1472 NR2B in the female rat hippocampus.

TL;DR: Reduction of pY1472 by estradiol suggests another mechanism through which estrogen enhances synaptic plasticity by altering localization of NMDAR subunits within synapses, as phosphorylation at Y1472 blocks NR2B endocytosis.