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Satoshi Suzuki

Researcher at Johns Hopkins University

Publications -  7
Citations -  963

Satoshi Suzuki is an academic researcher from Johns Hopkins University. The author has contributed to research in topics: Retina & Retinal. The author has an hindex of 6, co-authored 7 publications receiving 942 citations. Previous affiliations of Satoshi Suzuki include Johns Hopkins University School of Medicine.

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Journal ArticleDOI

Pattern electrical stimulation of the human retina

TL;DR: Experiments were conducted to study if electrical stimulation of the retinal surface can elicit visual sensation in individuals blind from end-stage retinitis pigmentosa (RP) or age-related macular degeneration (AMD).
Journal Article

Long-term histological and electrophysiological results of an inactive epiretinal electrode array implantation in dogs.

TL;DR: With the success of implanting an electrically inactive device onto the retinal surface for prolonged periods, the effects of long-term retinal electrical stimulation are now ready to be tested as the next step toward developing a prototype retinal prosthesis for human use.
Journal ArticleDOI

Comparison of electrical stimulation thresholds in normal and retinal degenerated mouse retina.

TL;DR: The amount of electrical charge required to elicit an action potential is dependent on the condition of the retina and the shape of the stimulus pulse used to deliver the charge.
Journal Article

Light-driven retinal ganglion cell responses in blind rd mice after neural retinal transplantation.

TL;DR: The presence of light-driven ganglion cell responses after subretinal transplantation in a retinal degenerate model demonstrates the presence of functional integration of the transplant with the host, but a rescue effect on remaining host photoreceptors cannot be ruled out.
Journal Article

Increased spontaneous retinal ganglion cell activity in rd mice after neural retinal transplantation.

TL;DR: Subretinal transplantation of neural retinal tissue results in a local increase of spontaneous ganglion cell activity, which may be due to the release of neurochemically active substances as a result of the presence of the transplant.