S
Satoshi Tani
Researcher at Kobe University
Publications - 22
Citations - 536
Satoshi Tani is an academic researcher from Kobe University. The author has contributed to research in topics: Cholecystokinin & Pancreatic juice. The author has an hindex of 11, co-authored 22 publications receiving 525 citations. Previous affiliations of Satoshi Tani include University of Occupational and Environmental Health Japan.
Papers
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Journal ArticleDOI
New model of acute necrotizing pancreatitis induced by excessive doses of arginine in rats.
Satoshi Tani,Hiroshi Itoh,Yoshinori Okabayashi,Takahiko Nakamura,Masatoshi Fujii,Takashi Fujisawa,Makoto Koide,Makoto Otsuki +7 more
TL;DR: It is demonstrated that the administration of excessive doses of arginine induces a new, noninvasive experimental model of acute necrotizing pancreatitis, and pancreatic architecture appeared almost normal after 14 days.
Journal ArticleDOI
Histologic and biochemical alterations in experimental acute pancreatitis induced by supramaximal caerulein stimulation
Satoshi Tani,Makoto Otsuki,Hiroshi Itoh,Masatoshi Fujii,Takahiko Nakamura,Toru Oka,Shigeaki Baba +6 more
TL;DR: Subcutaneous injections of 20 μg/kg body weight of caerulein induced a significant increase in serum amylase activity and histologic evidence of acute interstitial pancreatitis similar to those observed with the 50 μg/ kg body weight dosage of caERulein.
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Effect of Gymnema sylvestre, R.Br. on glucose homeostasis in rats
Yoshinori Okabayashi,Satoshi Tani,Takashi Fujisawa,Makoto Koide,Hiroshi Hasegawa,Takahiko Nakamura,Masatoshi Fujii,Makoto Otsuki +7 more
TL;DR: Results suggest the usefulness of G. sylvestre in the treatment of certain classes of non-insulin-dependent diabetes mellitus and show a tendency to reduce the serum glucose concentrations in the fed state and to improve the glucose tolerance.
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Effect of a new cholecystokinin receptor antagonist loxiglumide on acute pancreatitis in two experimental animal models.
Satoshi Tani,Yoshinori Okabayashi,Takahiko Nakamura,Masatoshi Fujii,Hiroshi Itoh,Makoto Otsuki +5 more
TL;DR: The results do not necessarily suggest that CCK is not important in the pathogenesis of pancreatitis, but do suggest that the sole blockade of peripheral CCK receptors isineffective against NaTc-induced severe necrotizing pancreatitis.
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Involvement of endogenous cholecystokinin in the development of acute pancreatitis induced by closed duodenal loop.
TL;DR: The observations suggest that endogenous CCK is not involved in the initiation of acute hemorrhagic and necrotizing pancreatitis induced by CDL, but is involvement in the development of pancreatitis in this model of acute pancreatitis.