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Scott H. Saul

Researcher at University of Pennsylvania

Publications -  39
Citations -  1674

Scott H. Saul is an academic researcher from University of Pennsylvania. The author has contributed to research in topics: Esophagus & Fibrolamellar hepatocellular carcinoma. The author has an hindex of 23, co-authored 39 publications receiving 1643 citations. Previous affiliations of Scott H. Saul include Hospital of the University of Pennsylvania.

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The Effect of a Pectin-Supplemented Elemental Diet on Intestinal Adaptation to Massive Small Bowel Resection

TL;DR: The effect of a pectin-supplemented elemental diet on intestinal adaptation to massive small bowel resection in the rat was investigated and mucosal parameters were significantly increased in the jejunum and ileum of both the resected pECTin and resecting no pect in this study.
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Barrett's metaplasia and adenocarcinoma of the esophagus in scleroderma

TL;DR: The records of 75 patients with scleroderma seen over a four-year period at the Hospital of the University of Pennsylvania were retrospectively reviewed to determine the prevalence of Barrett's metaplasia and adenocarcinoma of the esophagus and to identify clinical, manometric, laboratory, or radiographic criteria that might predict the presence of these lesions.
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Azathioprine and hepatic venocclusive disease in renal transplant patients.

TL;DR: It is suggested that azathioprine may be closely linked with the development of venocclusive disease in renal transplant patients and that the frequency of this disorder may be more common than previously reported.
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Blood Culture Positivity: Suppression by Outpatient Antibiotic Therapy in Patients With Bacterial Endocarditis

TL;DR: Patients with recent antibiotic exposure and suspected endocarditis should have blood cultures obtained initially and periodically throughout the phase of potential suppression of bacteremia to optimize the chance of obtaining a positive result.
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The immunohistochemistry of gastrointestinal stromal tumors. Evidence supporting an origin from smooth muscle.

TL;DR: The method of fixation, tissue preparation, and immunostaining may significantly affect the expression of desmin and most of these tumors show evidence of smooth muscle differentiation.