S
Seyed Ali Gaskari
Researcher at Tehran University of Medical Sciences
Publications - 15
Citations - 682
Seyed Ali Gaskari is an academic researcher from Tehran University of Medical Sciences. The author has contributed to research in topics: Cholestasis & Nitric oxide. The author has an hindex of 12, co-authored 15 publications receiving 645 citations. Previous affiliations of Seyed Ali Gaskari include University of Calgary.
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Journal ArticleDOI
Therapy insight: Cirrhotic cardiomyopathy.
TL;DR: The clinical features, general diagnostic criteria, pathogenesis and treatment of cirrhotic cardiomyopathy, which contributes to morbidity and mortality after liver transplantation, are discussed in this review.
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Role of endocannabinoids in the pathogenesis of cirrhotic cardiomyopathy in bile duct‐ligated rats
TL;DR: Results indicate a pathogenic role for increased local (neuronal) production of endocannabinoids, mediated by a Gi‐protein‐dependent CB‐1‐responsive pathway in cirrhotic cardiomyopathy, which may help explain why contractility is normal at baseline but attenuated with stress.
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The role of nitric oxide in anticonvulsant and proconvulsant effects of morphine in mice.
TL;DR: Results support the involvement of L-arginine/nitric oxide pathway in the modulation of seizure threshold by morphine.
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Anandamide mediates hyperdynamic circulation in cirrhotic rats via CB1 and VR1 receptors
Leila Moezi,Leila Moezi,Seyed Ali Gaskari,Hongqun Liu,Soon Koo Baik,Ahmad Reza Dehpour,Samuel S. Lee +6 more
TL;DR: The cardiovascular effects of anandamide, AM251 (CB1 antagonist), AM630 (CB2 antagonist) and capsazepine (VR1 antagonist) are examined in a rat model of cirrhosis to delineate the role of endocannabinoids in these changes.
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Mesenteric vascular bed responsiveness in bile duct-ligated rats: roles of opioid and nitric oxide systems
Khodadad Namiranian,Morteza Samini,Shahram Ejtemaei Mehr,Seyed Ali Gaskari,Hossein Rastegar,Houman Homayoun,Ahmad Reza Dehpour +6 more
TL;DR: Evidence is provided for the involvement of increased opioidergic tone and NO overproduction in cholestasis-induced vascular hyporesponsiveness and chronic treatment with L-NAME partially restored both the acetylcholine- induced vasorelaxation and phenylephrine-induced vasoconstriction response in bile duct-ligated rats.