S
Seyedhossein Aharinejad
Researcher at Medical University of Vienna
Publications - 140
Citations - 3910
Seyedhossein Aharinejad is an academic researcher from Medical University of Vienna. The author has contributed to research in topics: Vascular endothelial growth factor & Transplantation. The author has an hindex of 33, co-authored 140 publications receiving 3668 citations. Previous affiliations of Seyedhossein Aharinejad include University of Vienna & University of Massachusetts Medical School.
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Journal ArticleDOI
Rare case of high origin of the ulnar artery from the brachial artery
TL;DR: A high bifurcation of the brachial artery and the abnormal course of the ulnar artery is of interest to clinicians; in particular vascular and plastic surgeons and radiologists.
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The impact of colony‐stimulating factor‐1 on fracture healing: An experimental study
Kambiz Sarahrudi,Mehdi Mousavi,Karl Grossschmidt,Nezir Sela,Franz König,Vilmos Vécsei,Seyedhossein Aharinejad +6 more
TL;DR: Animals that were treated by CSF‐1 produced a significantly higher amount of mineralized bone over the first 8 weeks after fracture compared to the control animals, and a higher number of osteoclasts was found in CSf‐1‐treated animals within the first8 weeks, compared tothe controls.
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Low dose carboplatin combined with angiostatic agents prevents metastasis in human testicular germ cell tumor xenografts
TL;DR: Angiostatic agents added to low dose carboplatin have the ability to suppress the progression of human germ cell tumor xenografts toward a metastatic phenotype, indicating that this treatment strategy might be beneficial to prevent metastasis in germ cell tumors.
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New aspects of microvascular corrosion casting: a scanning, transmission electron, and high-resolution intravital video microscopic study.
TL;DR: The results show that undiluted Mercox is a stimulus for vascular cellular components and that it changes the vascular wall permeability, resulting in extravasation and filling of lymphatics and obtained casts are not fragile, as are casts of diluted Mercox, and phase separation does not occur.
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Frizzled2 signaling regulates growth of high-risk neuroblastomas by interfering with β-catenin-dependent and β-catenin-independent signaling pathways
Karin Zins,Romana Schäfer,Patrick Paulus,Silvia Dobler,Nazak Fakhari,Mouldy Sioud,Seyedhossein Aharinejad,Dietmar Abraham +7 more
TL;DR: A role for FZD2 in high-risk NB cell growth is suggested and a potential candidate for therapeutic inhibition in FzD2-expressing NB patients is provided.