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Shin Okamoto

Researcher at Kagoshima University

Publications -  85
Citations -  692

Shin Okamoto is an academic researcher from Kagoshima University. The author has contributed to research in topics: Quail & Genetic diversity. The author has an hindex of 13, co-authored 84 publications receiving 612 citations.

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Population Research of Genetic Polymorphism at Amino Acid Position 631 in Chicken Mx Protein with Differential Antiviral Activity

TL;DR: Investigation of whether various kinds of chicken breeds and jungle fowls carry the resistant or sensitive Mx allelic gene by using the mismatched PCR-restriction fragment length polymorphism (RFLP) technique confirmed the results by the determination of amino acid sequences in the GTPase effector domain of junglefowls.
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Genetic Diversity of mtDNA D-loop Polymorphisms in Laotian Native Fowl Populations

TL;DR: The results suggest that multiple maternal lineages were involved in the origin of domestic chicken in Laos, and there appear to be at least two maternal linesages, one from China and the other from the southeast Asian continent.
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Maturational changes in motility, acrosomal proteolytic activity, and penetrability of the inner perivitelline layer of fowl sperm, during their passage through the male genital tract

TL;DR: It is inferred that fowl sperm undergo a gradual process of maturational changes in motility, APA, and penetrability as a means of acquiring potential fertility during their passage throughout the male genital tract.
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Genetic Relationships Among Domestic and Jungle Fowls Revealed by DNA Fingerprinting Analysis

TL;DR: DFP analysis with mixed DNA could be effectively used to assess the genetic relationship among the stocks of fowls and indicated that Red Jungle Fowl was relatively close to the domestic fowlS.
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Indels within promoter and intron 1 of bovine prion protein gene modulate the gene expression levels in the medulla oblongata of two Japanese cattle breeds.

TL;DR: A significant correlation between promoter indel polymorphisms and PRNP-mRNA expression is found and it is hypothesized that differences in polymorphisms could be one of the causes of differences in PRNP expression levels.