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Shinji Kagami
Researcher at Oregon Health & Science University
Publications - 4
Citations - 990
Shinji Kagami is an academic researcher from Oregon Health & Science University. The author has contributed to research in topics: Corpus albicans & Antibody. The author has an hindex of 3, co-authored 4 publications receiving 904 citations.
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Journal ArticleDOI
Circulating Th17, Th22, and Th1 cells are increased in psoriasis.
Shinji Kagami,Heather L. Rizzo,Jennifer J. Lee,Yoshinobu Koguchi,Andrew Blauvelt,Andrew Blauvelt +5 more
TL;DR: Elevated numbers of circulating inflammatory T cells may contribute to cutaneous inflammation and systemic inflammatory disease that occurs in individuals with psoriasis.
Journal ArticleDOI
IL-23–Mediated Psoriasis-Like Epidermal Hyperplasia Is Dependent on IL-17A
Heather L. Rizzo,Shinji Kagami,Kevin G. Phillips,Stephen E. Kurtz,Steven L. Jacques,Andrew Blauvelt,Andrew Blauvelt +6 more
TL;DR: IL-17A, like IL-22, is a downstream mediator for IL-23–induced changes in murine skin and that both of these Th17 cytokines are necessary to produce IL- 23–mediated skin pathology.
Journal ArticleDOI
IL-23 and IL-17A, but Not IL-12 and IL-22, Are Required for Optimal Skin Host Defense against Candida albicans
Shinji Kagami,Heather L. Rizzo,Stephen E. Kurtz,Lloyd S. Miller,Andrew Blauvelt,Andrew Blauvelt +5 more
TL;DR: Exogenous recombinant IL-17A injected at the site of skin infection promoted more rapid healing of candidiasis in both wild-type mice and mice deficient in IL-23 and IL-12, and may serve as a potential therapy to enhance healing in individuals with chronic cutaneous candidiasis.
Hyperplasia Is Dependent on IL-17A Mediated Psoriasis-Like Epidermal - IL-23
TL;DR: This work presents a meta-analysis of immune checkpoint checkpoint checkpoints in rats over a 12-week period and shows clear patterns of decline in serum checkpoints over the course of a single vaccination.