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Shoichi Ozaki

Researcher at St. Marianna University School of Medicine

Publications -  123
Citations -  3538

Shoichi Ozaki is an academic researcher from St. Marianna University School of Medicine. The author has contributed to research in topics: Vasculitis & Microscopic polyangiitis. The author has an hindex of 32, co-authored 123 publications receiving 3213 citations. Previous affiliations of Shoichi Ozaki include Kyoto University & Juntendo University.

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A transgenic model of autoimmune hemolytic anemia.

TL;DR: This transgenic system providing a good autoimmune disease model for exploring its onset mechanism, and means of its treatment and prevention, and a wide variation of phenotypes between severe anemia and no symptoms.
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Intravenous cyclophosphamide therapy for progressive interstitial pneumonia in patients with polymyositis/dermatomyositis

TL;DR: In this open-label study, IVCYC improved symptoms, pulmonary function tests and HRCT findings in patients with PM/DM and longitudinal controlled studies are required to further confirm the efficacy and safety ofIVCYC.
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Isolation and characterization of CA XIV, a novel membrane-bound carbonic anhydrase from mouse kidney.

TL;DR: The isolation of a novel CA from the mouse kidney by use of the signal sequence trap method is succeeded and a functional cDNA encoding a novel membrane-bound CA is cloned, which is designated CA XIV.
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ANCA-associated vasculitis: diagnostic and therapeutic strategy.

TL;DR: The diagnostic criteria and the recent evidence-based therapeutic strategy of AAV are described and it is indicated that the rate of remission induction by the standard regimen is approximately 90% in 6 months, that maintenance of remission can be achieved with oral azathioprine as well as cyclophosphamide, and that methotrexate can be used only for non-renal mild AAV.
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Involvement of Th1 cells and heat shock protein 60 in the pathogenesis of intestinal Behcet's disease.

TL;DR: Th1‐dominant immune responses and HSP60 expression may induce the inflammatory responses and thus be associated with the pathogenesis of intestinal BD.