S
Simon Sanderson
Researcher at University of Cambridge
Publications - 38
Citations - 4207
Simon Sanderson is an academic researcher from University of Cambridge. The author has contributed to research in topics: Population & Health care. The author has an hindex of 17, co-authored 38 publications receiving 3912 citations.
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Tools for assessing quality and susceptibility to bias in observational studies in epidemiology: a systematic review and annotated bibliography
TL;DR: A number of useful assessment tools have been identified and a need to agree on critical elements for assessing susceptibility to bias in observational epidemiology and to develop appropriate evaluation tools.
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Effectiveness of physical activity promotion based in primary care: systematic review and meta-analysis of randomised controlled trials
TL;DR: Promotion of physical activity to sedentary adults recruited in primary care significantly increases physical activity levels at 12 months, as measured by self report, and there is insufficient evidence to recommend exercise referral schemes over advice or counselling interventions.
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CYP2C9 gene variants, drug dose, and bleeding risk in warfarin-treated patients: A HuGEnet™ systematic review and meta-analysis
TL;DR: Patients with CYP2C9*2 and CYP1C89*3 alleles have lower mean daily warfarin doses and a greater risk of bleeding, and testing for gene variants could potentially alter clinical management in patients commencing warfarine-treated patients.
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Obstacles and opportunities in meta-analysis of genetic association studies.
TL;DR: In this review, the impacts of Type I error, lack of power, and publication and reporting biases are explored, and the role of multiple testing is discussed.
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Thromboembolic events among adult patients with primary immune thrombocytopenia in the United Kingdom General Practice Research Database
Ameet Sarpatwari,Dimitri Bennett,John Logie,Amit Shukla,Kathleen J. Beach,Adrian C. Newland,Simon Sanderson,Drew Provan +7 more
TL;DR: Patients with primary immune thrombocytopenia are at increased risk for venous thromboembolic events compared with patients without primary immuneThrombocytes topenia.