S
Simon Zhornitsky
Researcher at Yale University
Publications - 78
Citations - 1613
Simon Zhornitsky is an academic researcher from Yale University. The author has contributed to research in topics: Cognition & Craving. The author has an hindex of 18, co-authored 68 publications receiving 1166 citations. Previous affiliations of Simon Zhornitsky include Foothills Medical Centre & Université de Montréal.
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Journal ArticleDOI
Cannabidiol in Humans—The Quest for Therapeutic Targets
Simon Zhornitsky,Stéphane Potvin +1 more
TL;DR: Preliminary clinical trials suggest that high-dose oral CBD may exert a therapeutic effect for social anxiety disorder, insomnia and epilepsy, but also that it may cause mental sedation.
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Oral versus Long-Acting Injectable Antipsychotics in the Treatment of Schizophrenia and Special Populations at Risk for Treatment Nonadherence: A Systematic Review.
Simon Zhornitsky,Emmanuel Stip +1 more
TL;DR: The efficacy and tolerability of LAIs versus their oral equivalents in randomized and naturalistic studies are examined and it is suggested that LAIs reduce risk of relapse versus oral antipsychotics in schizophrenia outpatients when combined with quality psychosocial interventions.
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Cholesterol and markers of cholesterol turnover in multiple sclerosis : Relationship with disease outcomes
TL;DR: The data indicate that cholesterol and markers of cholesterol turnover have potential to be used clinically as biomarkers of disease activity and may even be implicated in the pathogenesis of MS.
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Depression in multiple sclerosis: a long-term longitudinal study.
Marcus W. Koch,Scott B. Patten,Sandy Berzins,Simon Zhornitsky,Jamie Greenfield,Winona Wall,Luanne M. Metz +6 more
TL;DR: Depression in MS is largely chronic, which suggests a different pathophysiology from that of the general population, and antidepressant use was associated with a greater risk of depression at follow-up.
Journal Article
Antipsychotic-induced changes in blood levels of leptin in schizophrenia: a meta-analysis.
TL;DR: A physiological role of leptin in AP-induced weight gain is supported because the most significant leptin increases were observed with APs inducing the most weight gain and because of the observed association between leptin increases and BMI changes.