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Simon Zhornitsky
Researcher at Yale University
Publications - 78
Citations - 1613
Simon Zhornitsky is an academic researcher from Yale University. The author has contributed to research in topics: Cognition & Craving. The author has an hindex of 18, co-authored 68 publications receiving 1166 citations. Previous affiliations of Simon Zhornitsky include Foothills Medical Centre & Université de Montréal.
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Acute effects of ketamine and esketamine on cognition in healthy subjects: A meta-analysis
Simon Zhornitsky,Valérie Tourjman,Julie Pelletier,Roxane Assaf,Chiang-Shan R. Li,Stéphane Potvin +5 more
TL;DR: In this article , a multivariate meta-analysis was performed, and effect sizes were estimated for eleven cognitive domains: attention, executive function, response inhibition, social cognition, speed of processing, verbal / language, verbal learning, verbal memory, visual learning & memory, visuospatial abilities, and working memory.
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The effects of androgen deprivation on working memory and quality of life in prostate cancer patients: The roles of hypothalamic connectivity
Shefali Jayantibhai Chaudhary,Simon Zhornitsky,Alicia Roy,C. Summers,Tim A. Ahles,Chiang-Shan R. Li,Herta H. Chao +6 more
TL;DR: Given the wide regional connectivity of the hypothalamus and its role in regulating cognition and behavior, the effects of ADT on hypothalamic resting state functional connectivity and their cognitive and clinical correlates are assessed.
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Perceived burdensomeness and neural responses to ostracism in the Cyberball task.
TL;DR: Emotional distress in ostracism may increase with PB, resulting in stronger neural responses to social pain and behavioral avoidance of social interactions, according to an fMRI study of 64 adults performing the Cyberball task.
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Problem drinking alters gray matter volume and food cue responses of the lateral orbitofrontal cortex.
TL;DR: Evidence is provided for the effects of problem drinking on the brain substrates of feeding, potentially shedding light on the neural mechanisms underlying energy deficits in at‐risk drinkers.
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Acute quetiapine dose-dependently exacerbates anhedonia induced by withdrawal from escalating doses of d-amphetamine.
TL;DR: Results show that acute treatment with clinically relevant doses of quetiapine for the treatment of schizophrenia may exacerbate anhedonia induced by amphetamine withdrawal, an effect that decreased progressively over the next three days.