S
Siva K. Panguluri
Researcher at University of South Florida
Publications - 46
Citations - 1576
Siva K. Panguluri is an academic researcher from University of South Florida. The author has contributed to research in topics: Hyperoxia & Regulation of gene expression. The author has an hindex of 19, co-authored 44 publications receiving 1193 citations. Previous affiliations of Siva K. Panguluri include University of Kentucky & Indian Agricultural Research Institute.
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Journal ArticleDOI
Cardiovascular Risks Associated with Gender and Aging.
Jennifer L. Rodgers,Jarrod Jones,Samuel Ignatious Bolleddu,Sahit Vanthenapalli,Lydia E. Rodgers,Kinjal Shah,Krishna Karia,Siva K. Panguluri +7 more
TL;DR: Current findings regarding the impacts of age and gender on heart disease are discussed and hormone replacement therapies are largely shown to not improve outcomes in older patients and may also increase the risks of cardiac events in older adults.
Journal ArticleDOI
Targeted ablation of TRAF6 inhibits skeletal muscle wasting in mice
Pradyut K. Paul,Sanjay Kumar Gupta,Shephali Bhatnagar,Siva K. Panguluri,Bryant G. Darnay,Yongwon Choi,Ashok Kumar +6 more
TL;DR: TRAF6 expression is enhanced during muscle atrophy and induces activation of signal transduction cascades that promote muscle wasting.
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Isolation, characterization and expression of a novel vegetative insecticidal protein gene of Bacillus thuringiensis
R. Bhalla,Monika Dalal,Siva K. Panguluri,Borra Jagadish,Ajin Mandaokar,Amit Singh,Polumetla Ananda Kumar +6 more
TL;DR: The deduced amino acid sequence of the vip3Aa14 gene from Bacillus thuringiensis tolworthi showed considerable differences as compared to those of Vips reported so far, which were highly toxic to the larvae of Spodoptera litura and Plutella xylostella.
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Tumor necrosis factor-α regulates distinct molecular pathways and gene networks in cultured skeletal muscle cells.
Shephali Bhatnagar,Siva K. Panguluri,Sanjay Kumar Gupta,Saurabh Dahiya,Robert F. Lundy,Ashok Kumar +5 more
TL;DR: The study suggests that TNF-α activates both canonical and alternative NF-κB signaling pathways in a time-dependent manner in skeletal muscle cells, providing novel insight into the mechanisms of action of TNF.
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Genomic profiling of messenger RNAs and microRNAs reveals potential mechanisms of TWEAK-induced skeletal muscle wasting in mice.
Siva K. Panguluri,Shephali Bhatnagar,Akhilesh Kumar,John J. McCarthy,Apurva K. Srivastava,Nigel G. F. Cooper,Robert F. Lundy,Ashok Kumar +7 more
TL;DR: The results suggest that TWEAK affects the expression of several genes and microRNAs involved in inflammatory response, fibrosis, extracellular matrix remodeling, and proteolytic degradation which might be responsible for T WEAK-induced skeletal muscle loss.