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Stuart D. Saal

Researcher at NewYork–Presbyterian Hospital

Publications -  62
Citations -  2200

Stuart D. Saal is an academic researcher from NewYork–Presbyterian Hospital. The author has contributed to research in topics: Transplantation & Cholesterol. The author has an hindex of 24, co-authored 62 publications receiving 2125 citations. Previous affiliations of Stuart D. Saal include Columbia University Medical Center & Case Western Reserve University.

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Accumulation of normeperidine, an active metabolite of meperidine, in patients with renal failure of cancer

TL;DR: Data indicate that normeperidine can contribute to the excitatory effects seen after multiple doses of meperidine and suggest that patients with renal failure are particularly susceptible to this problem.
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Reversal of vascular and renal crises of scleroderma by oral angiotensin-converting-enzyme blockade.

TL;DR: Two cases of dramatic reversal of scleroderma vascular and renal crises with the oral angiotensin-converting-enzyme inhibitor SQ 14225 (Captopril) are reported.
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Low lipid concentrations in critical illness: implications for preventing and treating endotoxemia

TL;DR: Correcting the hypolipidemia by a reconstituted high-density lipoprotein preparation offers a new strategy for the prevention and treatment of endotoxemia.
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Relationship of hypolipidemia to cytokine concentrations and outcomes in critically ill surgical patients.

TL;DR: Low cholesterol and lipoprotein concentrations found in critically ill surgical patients correlate with interleukin-6, soluble interleukesin-2 receptor, and interleukain-10 concentrations and predict clinical outcomes.
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A single intravenous dose of endotoxin rapidly alters serum lipoproteins and lipid transfer proteins in normal volunteers.

TL;DR: A decline in LDL was rapidly induced in normal volunteers with a single iv dose of endotoxin, and the selective loss of phospholipid from HDL may have been mediated by LBP and, after more intense or prolonged inflammation, could result in increased HDL clearance and reduced HDL levels.