S
Sunil Yadav
Researcher at University of Miami
Publications - 39
Citations - 473
Sunil Yadav is an academic researcher from University of Miami. The author has contributed to research in topics: Myosin & Ricci curvature. The author has an hindex of 10, co-authored 32 publications receiving 321 citations. Previous affiliations of Sunil Yadav include University of Delhi & Coppin State University.
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Journal ArticleDOI
Fabrication, Optimization and Characterization of Natural Dye Sensitized Solar Cell.
William Ghann,Hyeonggon Kang,Tajbik Sheikh,Sunil Yadav,Tulio Chavez-Gil,Fred L. Nesbitt,Jamal Uddin +6 more
TL;DR: The dyes extracted from pomegranate and berry fruits were successfully used in the fabrication of natural dye sensitized solar cells (NDSSC) and showed a relatively low charge-transfer resistance and a long lifetime, signifying a reduction in recombination losses.
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Sarcomeric perturbations of myosin motors lead to dilated cardiomyopathy in genetically modified MYL2 mice.
Chen-Ching Yuan,Katarzyna Kazmierczak,Jingsheng Liang,Zhiqun Zhou,Sunil Yadav,Aldrin V. Gomes,Thomas C. Irving,Danuta Szczesna-Cordary +7 more
TL;DR: The data show that hypocontractile myosin motors and structural perturbations at the level of sarcomeres trigger aberrant functional remodeling in D94A hearts and the development of DCM, which closely follows the clinical phenotype and suggests that MYL2 may serve as a new therapeutic target for dilated cardiomyopathy.
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Phosphomimetic-mediated in vitro rescue of hypertrophic cardiomyopathy linked to R58Q mutation in myosin regulatory light chain.
TL;DR: S15D‐phosphomimic can be used as a potential rescue strategy to abrogate/alleviate the RLC mutation‐induced phenotypes and is a likely candidate for therapeutic intervention in HCM patients.
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Insights into myosin regulatory and essential light chains: a focus on their roles in cardiac and skeletal muscle function, development and disease.
TL;DR: This review article focuses on the involvement of myosin regulatory (RLC) and essential (ELC) light chains in striated muscle development, isoform appearance and their function in normal and diseased muscle, with a special focus on the role of myOSin II light chain (MLC) components.
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Hereditary heart disease: pathophysiology, clinical presentation, and animal models of HCM, RCM, and DCM associated with mutations in cardiac myosin light chains.
TL;DR: This review attempts to provide an overview of inherited cardiomyopathies with a focus on their genetic etiology in myosin regulatory (RLC) and essential (ELC) light chains, which are EF-hand protein family members with important structural and regulatory roles.