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Svetlana A. Ivanova

Researcher at Russian Academy of Sciences

Publications -  180
Citations -  1981

Svetlana A. Ivanova is an academic researcher from Russian Academy of Sciences. The author has contributed to research in topics: Tardive dyskinesia & Schizophrenia. The author has an hindex of 19, co-authored 169 publications receiving 1465 citations. Previous affiliations of Svetlana A. Ivanova include Siberian State Medical University & Mental Health Research Institute.

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Circuits regulating pleasure and happiness: the evolution of reward-seeking and misery-fleeing behavioral mechanisms in vertebrates.

TL;DR: Identifying the human correlate of the lamprey habenula-projecting globus pallidus may help in elucidating the mechanism of the antidepressant effects of glutamatergic drugs.
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Use of Carnosine for Oxidative Stress Reduction in Different Pathologies.

TL;DR: Special attention is paid to issues of use of carnosine in neurologic and mental diseases, in alcoholism as well as in physiological states accompanied by activation of free-radical processes and formation of oxidative stress.
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New insights into the mechanism of drug-induced dyskinesia.

TL;DR: Evidence is discussed that supports the hypothesis that indirect pathway MSNs are damaged in dyskinesia, which is an extrapyramidal movement disorder characterized by involuntary, repetitive, irregular motions that affect the mouth and face and/or the limbs and trunk.
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The role of the habenula in the transition from reward to misery in substance use and mood disorders

TL;DR: The habenula is divided into two anatomically and functionally distinct nuclei, the lateral (LHb) and the medial (MHb), which are primarily involved in reward-seeking and misery-fleeing behavior by controlling the RMTg and IPN, respectively.
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Circuits Regulating Pleasure and Happiness-Mechanisms of Depression.

TL;DR: This paper describes five theories in relationship to the model of the regulation of reward-seeking vs. misery-avoiding behaviors and suggests that reversal or compensation of these neuroplastic adjustments may explain the effects of biological therapies in treating depression.