scispace - formally typeset
T

Takahiro Suzuki

Researcher at Tohoku University

Publications -  72
Citations -  951

Takahiro Suzuki is an academic researcher from Tohoku University. The author has contributed to research in topics: Motion control & Computer science. The author has an hindex of 13, co-authored 68 publications receiving 769 citations. Previous affiliations of Takahiro Suzuki include Columbia University.

Papers
More filters
Journal ArticleDOI

Three-Dimensional Printed Multiphase Scaffolds for Regeneration of Periodontium Complex

TL;DR: A single stem/progenitor cell population appears to differentiate into putative dentin/cementum, PDL, and alveolar bone complex by scaffold's biophysical properties and spatially released bioactive cues for integrated periodontium regeneration.
Journal ArticleDOI

Regenerative Endodontics: Barriers and Strategies for Clinical Translation

TL;DR: Recent work using novel biomaterial scaffolds and growth factors that orchestrate the homing of host endogenous cells represents a departure from traditional cell transplantation approaches and may accelerate clinical translation in regeneration endodontics.
Journal ArticleDOI

Effects of growth factors on dental stem/progenitor cells

TL;DR: This review synthesizes knowledge on many growth factors that are known or anticipated to be efficacious in dental pulp-dentin regeneration to show that growth factor delivery, without cell transplantation, can yield pulp- dentin-like tissues in vivo.
Journal ArticleDOI

Resorbable poly-L-lactide plates and screws for the treatment of mandibular condylar process fractures: a clinical and radiologic follow-up study.

TL;DR: The PLLA miniplate system provides reliable stability when used for the fixation of mandibular condylar process fractures, and shows anatomic good reduction and long-term stability with the use of resorbable miniplates and screws.
Journal ArticleDOI

Puromycin-insensitive leucyl-specific aminopeptidase (PILSAP) binds and catalyzes PDK1, allowing VEGF-stimulated activation of S6K for endothelial cell proliferation and angiogenesis

TL;DR: It is demonstrated that PILSAP bound phosphatidylinositol-dependent kinase 1 (PDK1) and removed 9 amino acids from its N-terminus, which allowed S6K to associate with PDK1 and PilsAP upon VEGF stimulation, and the introduction of a mutated PILS AP gene in ECs inhibited angiogenesis and retarded tumor growth in vivo.