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Takashi Ueyama

Researcher at Wakayama Medical University

Publications -  82
Citations -  4789

Takashi Ueyama is an academic researcher from Wakayama Medical University. The author has contributed to research in topics: Cardiomyopathy & Estrogen. The author has an hindex of 27, co-authored 82 publications receiving 4040 citations.

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International Expert Consensus Document on Takotsubo Syndrome (Part I): Clinical Characteristics, Diagnostic Criteria, and Pathophysiology

TL;DR: The clinical expert consensus document part I summarizes the current state of knowledge on clinical presentation and characteristics of TTS and agrees on controversies surrounding TTS such as nomenclature, different TTS types, role of coronary artery disease, and etiology.
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Takotsubo Cardiomyopathy A New Form of Acute, Reversible Heart Failure

TL;DR: A condition featuring symptoms and signs of acute myocardial infarction without demonstrable coronary artery stenosis or spasm in which the heart takes on the appearance of a Japanese octopus fishing pot called a takotsubo is described and an animal model that may clarify the pathogenesis is discussed.
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International Expert Consensus Document on Takotsubo Syndrome (Part II): Diagnostic Workup, Outcome, and Management

TL;DR: The clinical expert consensus statement on takotsubo syndrome (TTS) part II focuses on the diagnostic workup, outcome, and management and summarizes the diagnostic approach, which may facilitate correct and timely diagnosis.
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Emotional stress induces transient left ventricular hypocontraction in the rat via activation of cardiac adrenoceptors: a possible animal model of 'tako-tsubo' cardiomyopathy.

TL;DR: Left ventriculography of rats experiencing emotional stress induced reversible LV apical ballooning, which was normalized by pretreatment with adrenoceptor blockade, supports activation of cardiac adrenoceptors in the absence of ischemia-reperfusion as the primary cause of this syndrome.
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Sequential mRNA expression for immediate early genes, cytokines, and neurotrophins in spinal cord injury

TL;DR: Of further interest was the finding that this upregulation of IL-1 beta, BDNF, NT-3, p75LNGFR and Trk B mRNAs in injured spinal cord was attenuated by treatment with high dose glucocorticoids, with the suggestion that the downregulation of BDNF andNT-3 might be disadvantageous to survival and axonal sprouting of spinal neurons.