T
Teresa Santos-Mendoza
Researcher at CINVESTAV
Publications - 18
Citations - 835
Teresa Santos-Mendoza is an academic researcher from CINVESTAV. The author has contributed to research in topics: PDZ domain & Mycobacterium tuberculosis. The author has an hindex of 7, co-authored 14 publications receiving 728 citations.
Papers
More filters
Journal ArticleDOI
Antimicrobial peptides: general overview and clinical implications in human health and disease.
TL;DR: This review focuses on cathelicin and defensins, the most documented human AMPs, and discusses their antimicrobial activity and pleiotropic immunomodulating effects on inflammatory and infectious diseases.
Journal ArticleDOI
Cellular and Humoral Mechanisms Involved in the Control of Tuberculosis
Joaquín Zúñiga,Diana Torres-García,Teresa Santos-Mendoza,Tatiana S. Rodriguez-Reyna,Julio Granados,Edmond J. Yunis +5 more
TL;DR: In this paper, the role of nonspecific inflammatory responses mediated by cytokines and chemokines induced by interaction of innate receptors expressed in macrophages and dendritic cells (DCs) was discussed.
Journal ArticleDOI
Seasonal and pandemic influenza H1N1 viruses induce differential expression of SOCS-1 and RIG-I genes and cytokine/chemokine production in macrophages
Gustavo Ramírez-Martínez,Alfredo Cruz-Lagunas,Luis Jiménez-Alvarez,Enrique Espinosa,Blanca Ortiz-Quintero,Teresa Santos-Mendoza,María Teresa Herrera,Elsy B. Canché-Pool,Criselda Mendoza,José Luis Bañales,Sara A. García-Moreno,Juan Morán,Carlos Cabello,Lorena Orozco,Irma Aguilar-Delfín,Alfredo Hidalgo-Miranda,Sandra Romero,Benjamin T. Suratt,Moisés Selman,Joaquín Zúñiga +19 more
TL;DR: Findings suggest that factors inherent to the pdm A/H1N1 viral strain may increase the production of inflammatory mediators by inhibiting SOCS-1 and modifying the expression of antiviral immunity-related genes, including RIG-I, in human macrophages.
Journal ArticleDOI
Diacylglycerol kinase ζ: at the crossroads of lipid signaling and protein complex organization.
TL;DR: The data suggest that DGKζ offers new opportunities for therapeutic manipulation of lipid metabolism, as well as demonstrating its subtle regulation and its strategic function in specific signaling and as part of adaptor protein complexes.
Journal ArticleDOI
Viral targeting of PDZ polarity proteins in the immune system as a potential evasion mechanism.
TL;DR: It is hypothesized that the viral PDZ‐dependent targeting of PDZ polarity proteins in these cells may alter the cellular fitness of the host to favor that of the virus, and occurs as a common and widespread mechanism for immune evasion by viruses and possibly other pathogens.