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Tommy Andersson

Researcher at Lund University

Publications -  210
Citations -  7330

Tommy Andersson is an academic researcher from Lund University. The author has contributed to research in topics: Wnt signaling pathway & Signal transduction. The author has an hindex of 45, co-authored 207 publications receiving 6598 citations. Previous affiliations of Tommy Andersson include Malmö University & Stockholm School of Economics.

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All-cause mortality in 272 186 patients hospitalized with incident atrial fibrillation 1995–2008: a Swedish nationwide long-term case–control study

TL;DR: Atrial fibrillation was an independent risk factor of all-cause mortality in patients with incident AF and the concomitant diseases that contributed most were found outside the thromboembolic risk scores.
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Wnt-5a has tumor suppressor activity in thyroid carcinoma.

TL;DR: Testing the effects of a member of the noncanonical Wnt signaling pathway, Wnt-5a, in primary thyroid carcinomas and in thyroid carcinoma cell lines found that a bell-shaped response was observed with low to undetectable levels in normal tissue and in anaplastic tumors whereas differentiated thyroid carcinomes showed strong positive immunostaining for Wnt -5a.
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WNT5A induces release of exosomes containing pro-angiogenic and immunosuppressive factors from malignant melanoma cells

TL;DR: Data indicate that WNT5A has a broader function on tumor progression and metastatic spread than previously known; by inducing exosome-release of immunomodulatory and pro-angiogenic factors that enhance the immunosuppressive and angiogenic capacity of the tumors thus rendering them more aggressive and more prone to metastasize.
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p38-MAPK Signals Survival by Phosphorylation of Caspase-8 and Caspase-3 in Human Neutrophils

TL;DR: In this paper, the p38-mitogen-activated protein kinase (MAPK) associates to caspase-8 and caspases-3 during neutrophil apoptosis.
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Loss of Wnt-5a protein is associated with early relapse in invasive ductal breast carcinomas.

TL;DR: Findings support the notion that this Wnt-5a protein retains tumor suppressor function by virtue of its effects on cell adhesion and motility in recurrent ductal breast cancer.