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Toshiyuki Yamamoto

Researcher at Fukushima Medical University

Publications -  681
Citations -  8596

Toshiyuki Yamamoto is an academic researcher from Fukushima Medical University. The author has contributed to research in topics: Medicine & Psoriasis. The author has an hindex of 42, co-authored 598 publications receiving 7643 citations. Previous affiliations of Toshiyuki Yamamoto include University of Cologne & Tokyo Medical and Dental University.

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Childhood tinea incognito caused by Trichophyton mentagrophytes var. interdigitale mimicking pustular psoriasis.

Yoshio Kawakami, +5 more
- 01 Nov 2011 - 
TL;DR: A case of pustular psoriasis‐like tinea incognito caused by Trichophyton mentagrophytes var.
Journal Article

[Dysprosody associated with environmental auditory sound agnosia in right temporal lobe hypoperfusion--a case report].

Toshiyuki Yamamoto, +5 more
- 01 Jan 2004 - 
TL;DR: It is considered that the lesion responsible for environmental auditory sound agnosia was present in the area around the secondary auditory area of the right temporal lobe and this patient differed from slowly progressive aphasia characterized by decreased blood flow in the left temporal lobe.
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Severe extra-palmoplantar pustulation associated with palmoplantar pustulosis.

Toshiyuki Yamamoto, +1 more
- 01 Aug 2004 - 
TL;DR: A case of PPP with severe extra-palmoplantar pustulation, for which systemic cyclosporin resulted in beneficial effects is described, which is frequently resistant to most forms of topical treatment.
Journal ArticleDOI

Coexistence of subepidermal calcified nodule and keratosis punctata in a patient with hyperhidrosis.

Toshiyuki Yamamoto, +2 more
- 01 Jun 1995 - 
TL;DR: A 47‐year‐old female with hyperhidrosis developed multiple subepidermal calcified nodules and keratosis punctata on her palms, and it was supposed that the eccrine sweat glands may have played a role in inducing these two lesions.
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Pancreatic developmental defect evaluated by celiac artery angiography in a patient with MODY5

Naoko Iwasaki, +9 more
- 14 Jul 2016 - 
TL;DR: A disruption of the great pancreatic artery is identified in a patient with MODY5 with no pancreatic body or tail, which supports the significance of HNF1B in the development of the pancreas.