Tracy J. Abildskov

TL;DR: The findings suggest that long-range connectivity abnormalities in autism are spatially heterogeneous and that transcallosal connectivity is decreased most in regions with functions associated with behavioral abnormalities in Autism.

TL;DR: It is suggested that abnormal cortical development in autism spectrum disorders undergoes three distinct phases: accelerated expansion in early childhood, acceleratedthinning in later childhood and adolescence, and decelerated thinning in early adulthood.

TL;DR: WMHs, hemosiderin deposits, and focal areas of encephalomalacia or atrophy were common in children with TBI, were related to injury severity, and were mostly observed within a frontotemporal distribution.

TL;DR: Conative ToM was significantly and positively related to the package of Default Mode, Central Executive, and Mirror Neuron Empathy networks and, more specifically, to two hubs of the Default Mode Network, the posterior cingulate/retrosplenial cortex and the hippocampal formation, including entorhinal cortex and parahippocampal cortex.

TL;DR: It is suggested that VM-PFD may be associated with a wider spectrum of cognitive deficits than previously characterized, and C.D. appeared to sacrifice accuracy for speed and to adopt liberal response strategies, implicating problems with cognitive inflexibility, impulsivity, and disinhibition.