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Ursula B. Kaiser
Researcher at Brigham and Women's Hospital
Publications - 232
Citations - 16273
Ursula B. Kaiser is an academic researcher from Brigham and Women's Hospital. The author has contributed to research in topics: Gonadotropin-releasing hormone & Receptor. The author has an hindex of 62, co-authored 210 publications receiving 14115 citations. Previous affiliations of Ursula B. Kaiser include University of Pennsylvania & St. Michael's Hospital.
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Journal ArticleDOI
Editorial: The Rise of the Asterisk: One Step to Facilitate Team Science.
TL;DR: It appears that investigators, at least in the field of endocrinology, have recognized and acted upon the need for collaborative efforts in order to most effectively advance scientific discoveries even before the research community as a whole began to recognize and promote this need.
Journal ArticleDOI
Connecting nutritional deprivation and pubertal inhibition via GRK2-mediated repression of kisspeptin actions in GnRH neurons.
Cecilia Perdices-Lopez,María S. Avendaño,Alexia Barroso,F Gaytan,Francisco Ruiz-Pino,María J. Vázquez,Silvia Leon,Yong Bhum Song,Verónica Sobrino,Violeta Heras,Antonio Romero-Ruiz,Juan Carlos Roa,Federico Mayor,Cristina Murga,Leonor Pinilla,Ursula B. Kaiser,Manuel Tena-Sempere +16 more
TL;DR: In this paper , the putative role of the G protein-coupled-receptor kinase-2, GRK2, in GnRH neurons, as modulator of pubertal timing via repression of the actions of kisspeptin, in normal maturation and conditions of nutritional deficiency, was addressed.
An activating mutation in GPR54 gene causes gonadotropin-dependent precocious puberty
Milena Gurgel Teles,Vinicius Nahime Brito,Suzy C. Bianco,Ericka B. Trarbach,Stephanie B. Seminara,Ivo J.P. Arnhold,Ursula B. Kaiser,Ana Claudia Latronico,Berenice B. Mendonca +8 more
Journal Article
Persistent hyperkalemia in a patient with diabetes mellitus: a reversible defect in kaliuresis during bicarbonaturia.
TL;DR: Exretion of potassium was low, considering the stimulus of hyperkalemia, and did not rise appreciably after the acute or chronic administration of a mineralocorticoid, and it is speculated that the basis for the hyperKalemia was type II hypoaldosteronism.
Journal ArticleDOI
Hypothalamic miR-30 regulates puberty onset via repression of the puberty-suppressing factor, Mkrn3
Juan M. Castellano,Violeta Heras,Susana Sangiao-Alvarellos,Maria Manfredi-Lozano,María J. Sanchez-Tapia,Francisco Ruiz-Pino,Juan Carlos Roa,Maribel Lara-Chica,Rosario Morrugares-Carmona,Ana Paula Abreu,Denise D. Belsham,María J. Vázquez,Marco A. Calzado,Leonor Pinilla,Francisco Gaytan,Ana Claudia Latronico,Ursula B. Kaiser,Manuel Tena-Sempere +17 more
TL;DR: It is reported that the microRNA, miR-30, with three binding sites in a highly conserved region of its 3 UTR, operates as repressor of Mkrn3 to control pubertal onset and expands the current understanding of the molecular basis of puberty and its disease states.