Vishal Saxena

TL;DR: Peripheral blood microscopy, parasite antigen–based assays, and parasite 18s rRNA gene–based polymerase chain reaction showed the presence of P. vivax and absence ofP.

TL;DR: Among patients with severe manifestations, 40 had evidence of monoinfection of P. vivax malaria, and further large-scale multicentric epidemiologic studies are needed to define the basic pathology of this less known entity.

TL;DR: The evidence of severe P. vivax malaria in children in Bikaner is reaffirmed and the proportion of patients having severe manifestations, which included severe anemia, thrombocytopenia, cerebral malaria, acute respiratory distress syndrome, hepatic dysfunction, renal dysfunction, abnormal bleeding was significantly high in association with P. falciparum.

TL;DR: It was found that the association of thrombocytopenia was statistically more significant with P. vivax monoinfection as compared to P. falciparum.

TL;DR: In vitro phenotyping and RNA-seq analyses revealed key microenvironmental relationships with distinct biological functions and preserved sporozoite quiescence by exposure to insect-like factors coupled with strategic activation limits untimely activation of invasion-associated genes to dramatically increase hepatocyte invasion rates.