V
Vsevolod Zinserling
Publications - 10
Citations - 419
Vsevolod Zinserling is an academic researcher. The author has contributed to research in topics: Medicine & Internal medicine. The author has an hindex of 1, co-authored 1 publications receiving 176 citations.
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Journal ArticleDOI
Covid-19 and autoimmunity.
Michael Ehrenfeld,Angela Tincani,Laura Andreoli,Marco Cattalini,Assaf Greenbaum,Darja Kanduc,Jaume Alijotas-Reig,Vsevolod Zinserling,Natalia Semenova,Howard Amital,Yehuda Shoenfeld +10 more
TL;DR: A new strain of coronavirus infection emerged in Wuhan, China the SARS-CoV-2 or the Covid-19 as mentioned in this paper, which is characterized by fever, dry cough, myalgia and extreme fatigue.
Journal ArticleDOI
Neutrophils Mediate Pulmonary Artery Thrombosis In Situ
O. Porembskaya,Vsevolod Zinserling,V. V. Tomson,Ya. G. Toropova,Eleonora A. Starikova,Vitaliy V. Maslei,Nika Bulavinova,O. V. Kirik,M. A. Syrtsova,Leonid Laberko,M. I. Galchenko,V. Kravchuk,Sergey Saiganov,Alexander Brill +13 more
TL;DR: It is demonstrated that thrombi in the lung vasculature can form in situ by mechanisms that require local neutrophil recruitment taking place in the DVT setting.
Journal ArticleDOI
Infections and schizophrenia
TL;DR: A critical review of the literature demonstrates a certain pathogenetic role of various infections, primarily viruses from the herpes and chlamydia groups, in the development and progression of schizophrenia, including published results of the authors’ own long-term studies.
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Case of successful treatment of disseminated pneumocystosis with lesions of lungs and small intestine in an HIV-infected patient
TL;DR: In this article , a rare case of pneumocystosis of the small intestine of a 39-year-old man with HIV infection using histological and immunohistochemical methods was described.
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SARS-CoV-2-Induced Pathology—Relevance to COVID-19 Pathophysiology
Vsevolod Zinserling,Natalia Semenova,Anastasia E. Bikmurzina,Natalia M. Kruglova,Oksana V. Rybalchenko,Alexander G. Markov +5 more
TL;DR: The results of the histological and IHC studies of deceased people with varying degrees of severity of coronavirus infection confirmed the ability of these pathogens to cause cytoproliferative changes, primarily in epithelial and endothelial cells.