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W. Gerald Austen
Researcher at Harvard University
Publications - 173
Citations - 6522
W. Gerald Austen is an academic researcher from Harvard University. The author has contributed to research in topics: Cardiogenic shock & Myocardial infarction. The author has an hindex of 48, co-authored 173 publications receiving 6459 citations. Previous affiliations of W. Gerald Austen include National Institutes of Health & Brown University.
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Journal ArticleDOI
Immunological release of histamine and slow-reacting substance of anaphylaxis from human lung : I. Modulation by agents influencing cellular levels of cyclic 3',5'-adenosine monophosphate.
TL;DR: In this article, drugs capable of increasing cellular levels of CAMP such as beta-adrenergic agents and methylxanthines inhibit the antigen-induced release of both histamine and SRS-A from human lung and these agents demonstrate synergism when used together.
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Clinical and Hemodynamic Results of Intraaortic Balloon Pumping and Surgery for Cardiogenic Shock
W. Bruce Dunkman,Robert C. Leinbach,Mortimer J. Buckley,Eldred D. Mundth,Arthur Kantrowitz,W. Gerald Austen,Charles A. Sanders +6 more
TL;DR: IABP is a safe, effective means of supporting the circulation in CS-MI, and IABP alone will improve survival in some patients, and some patients with CS- MI have myocardial necrosis too extensive to permit survival without permanent circulatory assistance or total cardiac replacement.
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Long-term survival of patients with treated aortic dissection
Robert M. Doroghazi,Eve E. Slater,Roman W. DeSanctis,Mortimer J. Buckley,W. Gerald Austen,Simon V. Rosenthal +5 more
TL;DR: Retrospective data on the treatment of aortic dissection at the Massachusetts General Hospital from 1963 to 1978 are reported, showing that the success of early definitive medical and surgical treatment was sustained on long-term follow-up.
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Acute traumatic disruption of the thoracic aorta: a ten-year experience.
TL;DR: Immediate operative intervention with a heparinized shunt is preferable as soon as the diagnosis of thoracic aortic disruption has been established, but elective delay of operation in patients with severe concomitant injuries can be achieved safely with beta blockade and antihypertensive therapy.
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Mitral valve reconstruction versus replacement for degenerative or ischemic mitral regurgitation.
Cary W. Akins,Alan D. Hilgenberg,Mortimer J. Buckley,Gus J. Vlahakes,David F. Torchiana,Willard M. Daggett,W. Gerald Austen +6 more
TL;DR: Mitral valve reconstruction, when technically feasible, is the procedure of choice for degenerative or ischemic mitral regurgitation because of significantly lower hospital mortality and late valve-related events.