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Walter Glender

Researcher at National Autonomous University of Mexico

Publications -  6
Citations -  132

Walter Glender is an academic researcher from National Autonomous University of Mexico. The author has contributed to research in topics: Nucleoside & Adenosine. The author has an hindex of 5, co-authored 6 publications receiving 130 citations. Previous affiliations of Walter Glender include Hospital General de México.

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Effects of adenosine on liver cell damage induced by carbon tetrachloride

TL;DR: The results suggest that the antilipolytic effect of the nucleoside, the inhibition of hepatic fatty acid metabolism, and the decrease in carbon tetrachloride-induced lipoperoxidation that it produced are involved in the delayed acute hepatotoxicity induced by carbon t trichloride.
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Circadian variations of adenosine level in blood and liver and its possible physiological significance

TL;DR: The results suggest that liver purine metabolism might participate in the maintenance and renewal of the blood purine pool and in the energy state of erythrocytes in vivo.
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Mycobacterium fortuitum glycolipids for the serodiagnosis of pulmonary tuberculosis.

TL;DR: Glycolipids belonging to the family of acylated trehaloses were isolated from Mycobacterium fortuitum, a rapidly growing mycobacterial species, and tested in the serologic diagnosis of human pulmonary tuberculosis by enzyme-linked immunosorbent assay.
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Effects of adenosine on ethanol-induced modifications of liver metabolism: Role of hepatic redox state, purine and fatty acid metabolism

TL;DR: In this article, a total prevention of ethanol-induced fatty liver by the simultaneous administration of adenosine and allopurinol was observed, showing that the nucleoside prevented fatty liver in the presence of allopuroninol and this was related to an inhibition of fatty acid metabolism.
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Alterations of ATP levels and of energy parameters in the blood of alcoholic and nonalcoholic patients with liver damage

TL;DR: Although the mechanism involved in these effects remains to be elucidated, a failure of the damaged liver to supply purines to extra-hepatic tissues might be a major event altering the blood energy parameters.