Wanchun Tang

TL;DR: In this healthy animal model, asphyxial cardiac arrest resulted in significantly lesser impairment of postresuscitation myocardial function when compared with cardiac arrest caused by VF.

TL;DR: Active compression-decompression cardiopulmonary resuscitation + an impedance threshold device increased cerebral perfusion pressures and lowered diastolic intrusion pressure and intracranial pressure rate during the decompression phase, which may underlie the observed increase in cerebral perfusions pressure, carotid blood flow, and survival rates with favorable neurologic outcomes.

TL;DR: Increases in Pmco2 and [H+] were observed in the absence of increases in the total CO2 content and therefore the calculated myocardial bicarbonate, and these observations in the rat model implicate buffering of metabolic acids by biccarbonate rather than increases in CO2 production or decreases inCO2 removal as the predominant mechanism accounting for myocardia hypercarbia.

TL;DR: In this model of prolonged cardiac arrest, nasopharyngeal cooling initiated during cardiopulmonary resuscitation improved the success of resuscitation compared to cooling with cold saline infusion.

TL;DR: In this review, tissue PCO2 is identified as a universal indicator of impaired perfusion and contrasted with conventional hemodynamic and metabolic markers of perfusion failure.