W
Wayne Hing
Researcher at Bond University
Publications - 181
Citations - 4036
Wayne Hing is an academic researcher from Bond University. The author has contributed to research in topics: Medicine & Population. The author has an hindex of 31, co-authored 168 publications receiving 3293 citations. Previous affiliations of Wayne Hing include University of Birmingham & RMIT University.
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Journal ArticleDOI
Physiological response to water immersion: a method for sport recovery?
TL;DR: Water immersion may cause physiological changes within the body that could improve recovery from exercise and there may be a psychological benefit to athletes with a reduced cessation of fatigue during immersion.
Journal ArticleDOI
Neural Mobilization: A Systematic Review of Randomized Controlled Trials with an Analysis of Therapeutic Efficacy
Richard Ellis,Wayne Hing +1 more
TL;DR: A systematic review of the literature pertaining to the therapeutic efficacy of neural mobilization is provided in this paper, which highlights the lack in quantity and quality of the available research, and concludes that there is only limited evidence to support the use of neural mobilisation as a treatment modality.
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High-intensity interval training versus moderate-intensity continuous training within cardiac rehabilitation: a systematic review and meta-analysis
Amanda L Hannan,Wayne Hing,Vini Simas,Mike Climstein,Mike Climstein,Jeff S. Coombes,Rohan Jayasinghe,Rohan Jayasinghe,Joshua Byrnes,James Furness +9 more
TL;DR: HIIT appears to be as safe as MICT for CR participants, and improvements in cardiorespiratory fitness are significant for CR programs of >6-week duration.
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International framework for examination of the cervical region for potential of Cervical Arterial Dysfunction prior to Orthopaedic Manual Therapy intervention.
TL;DR: The purpose of the framework is to provide guidance to clinicians for the assessment of the cervical region for potential of Cervical Arterial Dysfunction in advance of planned management (inclusive of manual therapy and exercise interventions).
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Different effects of raised [K+]o on membrane potential and contraction in mouse fast- and slow-twitch muscle
TL;DR: It is suggested that the K(+)-depressed force is due to reduced Ca2+ release resulting from an altered action potential profile and inexcitable fibers due to an increased action potential threshold (phase 2), and K+ contributes to fatigue in both fast- and slow-twitch muscle when it causes depolarization to about -60 mV.