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Wen-Jun Wang
Researcher at Kunming University of Science and Technology
Publications - 7
Citations - 250
Wen-Jun Wang is an academic researcher from Kunming University of Science and Technology. The author has contributed to research in topics: Cell cycle & Cell growth. The author has an hindex of 6, co-authored 7 publications receiving 171 citations.
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Journal ArticleDOI
miR-145-5p Suppresses Tumor Cell Migration, Invasion and Epithelial to Mesenchymal Transition by Regulating the Sp1/NF-κB Signaling Pathway in Esophageal Squamous Cell Carcinoma.
TL;DR: The results suggested that miR-145-5p plays tumor-suppressive roles by inhibiting esophageal cancer cell migration, invasion and EMT through regulating the Sp1/NF-κB signaling pathway.
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Ferroptosis in Carcinoma: Regulatory Mechanisms and New Method for Cancer Therapy
Zhi-Zhou Shi,Ze-Wen Fan,Yun-Xia Chen,Xiu-Feng Xie,Wen Jiang,Wen-Jun Wang,Yun-Tan Qiu,Jie Bai +7 more
TL;DR: Recent findings on the regulatory mechanisms of key regulators of ferroptosis, including the catalytic subunit solute carrier family 7 member 11 (SLC7A11), the glutathione peroxidase 4 (GPX4), p53 and non-coding RNAs are summarized.
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miR-125b-5p functions as a tumor suppressor gene partially by regulating HMGA2 in esophageal squamous cell carcinoma.
TL;DR: The results suggested that overexpression of miR-125b-5p inhibited cell proliferation, migration and invasion partially by down-regulating HMGA2 in ESCC.
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MCM7 amplification and overexpression promote cell proliferation, colony formation and migration in esophageal squamous cell carcinoma by activating the AKT1/mTOR signaling pathway
TL;DR: It is suggested that MCM7 promoted tumor cell proliferation, colony formation and migration of ESCC cells via activating AKT1/mTOR signaling pathway.
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MiR-99a suppresses proliferation, migration and invasion of esophageal squamous cell carcinoma cells through inhibiting the IGF1R signaling pathway.
TL;DR: The findings suggested that loss of miR-99a in ESCC promoted the tumor cell proliferation, migration, invasion and slug-induced EMT through activating IGF1R signaling pathway.