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Wenqi Wu
Researcher at Guangzhou Medical University
Publications - 137
Citations - 5438
Wenqi Wu is an academic researcher from Guangzhou Medical University. The author has contributed to research in topics: Percutaneous nephrolithotomy & Medicine. The author has an hindex of 27, co-authored 121 publications receiving 4354 citations. Previous affiliations of Wenqi Wu include University of Duisburg-Essen.
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γ-H2AX in recognition and signaling of DNA double-strand breaks in the context of chromatin
TL;DR: The role of γ-H2AX in DNA damage response in the context of chromatin is reviewed and the use of this modification as a surrogate marker for mechanistic studies of DSB induction and processing is discussed.
SURVEY AND SUMMARY c-H2AX in recognition and signaling of DNA double-strand breaks in the context of chromatin
TL;DR: In this article, the role of the histone H2A variant, H2AX, in DNA double-strand break (DSBs) induction and processing has been discussed.
Journal ArticleDOI
PARP-1 and Ku compete for repair of DNA double strand breaks by distinct NHEJ pathways
TL;DR: It is shown that PARP-1 operates in an alternative pathway of non-homologous end joining (NHEJ) that functions as backup to the classical pathway of NHEJ that utilizes DNA-PKcs, Ku, DNA ligase IV, XRCC4, XLF/Cernunnos and Artemis.
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Prevalence of kidney stones in China: an ultrasonography based cross-sectional study
Guohua Zeng,Zanlin Mai,Shujie Xia,Zhiping Wang,Keqin Zhang,Li Wang,Yongfu Long,Jinxiang Ma,Yi Li,Show P. Wan,Wenqi Wu,Yongda Liu,Zelin Cui,Zhijian Zhao,Jing Qin,Tao Zeng,Yang Liu,Xiaolu Duan,Xin Mai,Zhou Yang,Zhenzhen Kong,Tao Zhang,Chao Cai,Yi Shao,Zhong-Jin Yue,Shujing Li,Jiandong Ding,Shan Tang,Zhangqun Ye +28 more
TL;DR: To investigate the prevalence and associated factors of kidney stones among adults in China, a large number of patients are diagnosed with at least one type of kidney stone each year.
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Repair of radiation induced DNA double strand breaks by backup NHEJ is enhanced in G2.
TL;DR: The results show a new and potentially important cell cycle regulation of B-NHEJ and generate a framework to investigate the mechanistic basis of HRR contribution to DSB repair.