W
Wim Van Camp
Researcher at Ghent University
Publications - 49
Citations - 5223
Wim Van Camp is an academic researcher from Ghent University. The author has contributed to research in topics: Acrylate & Copolymer. The author has an hindex of 29, co-authored 49 publications receiving 4997 citations. Previous affiliations of Wim Van Camp include Carnegie Mellon University & Plant Genetic Systems.
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Journal ArticleDOI
Catalase is a sink for H2O2 and is indispensable for stress defence in C3 plants.
Hilde Willekens,Sangpen Chamnongpol,Mark W. Davey,Martina Schraudner,Christian Langebartels,Marc Van Montagu,Dirk Inzé,Wim Van Camp +7 more
TL;DR: Leaf necrosis correlated with accumulation of oxidized glutathione and a 4‐fold decrease in ascorbate, indicating that catalase is critical for maintaining the redox balance during oxidative stress and may not be limited to peroxisomal H2O2 production.
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"Clicking" Polymers or Just Efficient Linking: What Is the Difference?**
Christopher Barner-Kowollik,Filip Du Prez,Pieter Espeel,Craig J. Hawker,Thomas Junkers,Helmut Schlaad,Wim Van Camp +6 more
TL;DR: In this paper, a set of click criteria are evaluated specifically for the synthetic polymer field, and the original click criteria were evaluated in an Essay specifically for synthetic polymer fields and the set of criteria are specified that distinguish click from other efficient reactions.
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Defense activation and enhanced pathogen tolerance induced by H2O2 in transgenic tobacco
Sangpen Chamnongpol,Hilde Willekens,Wolfgang Moeder,Christian Langebartels,Heinrich Sandermann,Marc Van Montagu,Dirk Inzé,Wim Van Camp +7 more
TL;DR: It is demonstrated that sublethal levels of H2O2 activate expression of acidic and basic PR proteins and lead to enhanced pathogen tolerance, however, rapid and strong activation of PR protein expression, as seen during the hypersensitive response, occurs only when excess H 2O2 is accompanied by leaf necrosis.
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Transgenic tobacco plants with reduced capability to detoxify reactive oxygen intermediates are hyperresponsive to pathogen infection.
Ron Mittler,Elza Hallak Herr,Bjorn Larus Orvar,Wim Van Camp,Hilde Willekens,Dirk Inzé,Brian E. Ellis +6 more
TL;DR: The hypothesis that suppression of ROI-scavenging enzymes during the hypersensitive response plays an important role in enhancing pathogen-induced PCD is supported.
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Ozone‐induced oxidative burst in the ozone biomonitor plant, tobacco Bel W3
Martina Schraudner,Wolfgang Moeder,Cosima B. Wiese,Wim Van Camp,Dirk Inzé,Christian Langebartels,Heinrich Sandermann +6 more
TL;DR: The results indicate that ozone effects are amplified in the sensitive tobacco cv.