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Xingduo Ji

Researcher at Chongqing Medical University

Publications -  7
Citations -  144

Xingduo Ji is an academic researcher from Chongqing Medical University. The author has contributed to research in topics: Biosensor & Offspring. The author has an hindex of 4, co-authored 6 publications receiving 82 citations.

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Journal ArticleDOI

Target-inspired Pb2+-dependent DNAzyme for ultrasensitive electrochemical sensor based on MoS2-AuPt nanocomposites and hemin/G-quadruplex DNAzyme as signal amplifier.

TL;DR: This proposed biosensor is ultrasensitive and specific, representing a potential application for the detection of Pb2+ in a water environment and exhibiting a lower detection limit of 38 fg mL-1 (based on 3σ).
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Dandelion-like CuO microspheres decorated with Au nanoparticle modified biosensor for Hg2+ detection using a T-Hg2+-T triggered hybridization chain reaction amplification strategy.

TL;DR: The proposed biosensor combined the amplification of the HCR and the inherent redox activity of TB and utilized D-CuO/Au composites, which exhibited high sensitivity for Hg2+ determination.
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Fabrication of pioneering 3D sakura-shaped metal-organic coordination polymers Cu@L-Glu phenomenal for signal amplification in highly sensitive detection of zearalenone

TL;DR: The proposed electrochemical aptasensor shows excellent selectivity to the ZEN in the presence of possible interfering substances, and has potential application for ZEN detection in food samples.
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Trimetallic signal amplification aptasensor for TSP-1 detection based on Ce-MOF@Au and AuPtRu nanocomposites

TL;DR: In this work, an aptamer was used as the target capturing agent and a trimetallic signal amplification strategy based on Ce-MOF@Au and AuPtRu NPs was demonstrated for the sensitive detection of TSP-1.
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In utero Exposure to Excessive Estrogen Impairs Homologous Recombination and Oogenesis via Estrogen Receptor 2 in Mice.

TL;DR: In this article, the authors found that in utero estrogen exposure can affect homologous recombination in early oogenesis and influence the reproductive potential and lifespan of female offspring, and that mid-gestation exposure to estrogen also led to delayed primordial folliculogenesis after birth, impaired follicle development after prepuberty and ultimately reduced the total litter size of the offspring.