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Y-D. Ida Chen

Researcher at Stanford University

Publications -  78
Citations -  8311

Y-D. Ida Chen is an academic researcher from Stanford University. The author has contributed to research in topics: Insulin & Diabetes mellitus. The author has an hindex of 45, co-authored 78 publications receiving 8120 citations. Previous affiliations of Y-D. Ida Chen include United States Department of Veterans Affairs & National Defense Medical Center.

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Journal ArticleDOI

Relationship Between Resistance to Insulin-Mediated Glucose Uptake, Urinary Uric Acid Clearance, and Plasma Uric Acid Concentration

TL;DR: Urinary uric acid clearance appears to decrease in proportion to increases in insulin resistance in normal volunteers, leading to an increase in serum uric Acid concentration, which appears to be exerted at the level of the kidney.
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Insulin resistance and cigarette smoking

TL;DR: The findings show that chronic cigarette smokers are insulin resistant, hyperinsulinaemic, and dyslipidaemic compared with a matched group of non-smokers, and may help to explain why smoking increases risk of coronary heart disease.
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Resistance to insulin-stimulated-glucose uptake in patients with hypertension.

TL;DR: Differences in the resultant SSPG concentrations allowed direct comparison of insulin’s ability to stimulate disposal of an glucose challenge and insulin-stimulated glucose uptake in Chinese men to be compared.
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A comparison between the minimal model and the glucose clamp in the assessment of insulin sensitivity across the spectrum of glucose tolerance. Insulin Resistance Atherosclerosis Study.

TL;DR: The insulin-modified frequently sampled intravenous glucose tolerance test (FSIGTT) with minimal model analysis provides estimates of insulin sensitivity that correlate significantly with those from the glucose clamp, however, in NIDDM.
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Effects of Varying Carbohydrate Content of Diet in Patients With Non—Insulin-Dependent Diabetes Mellitus

TL;DR: In NIDDM patients, high-carbohydrate diets compared with high-monounsaturated-fat diets caused persistent deterioration of glycemic control and accentuation of hyperinsulinemia, as well as increased plasma triglyceride and very-low-density lipoprotein cholesterol levels, which may not be desirable.