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Yali Jia

Researcher at Oregon Health & Science University

Publications -  240
Citations -  15683

Yali Jia is an academic researcher from Oregon Health & Science University. The author has contributed to research in topics: Optical coherence tomography & Glaucoma. The author has an hindex of 49, co-authored 236 publications receiving 13061 citations. Previous affiliations of Yali Jia include University of Washington.

Papers
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Journal ArticleDOI

Retinal capillary oximetry with visible light optical coherence tomography

TL;DR: In vivo assessment of hemoglobin oxygenation across the full vascular transition from retinal arteries to capillaries to retinal veins in rats is reported using visible-light optical coherence tomography and physiologic responses to alterations in the concentration of inhaled oxygen are demonstrated.
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Reduced Retinal Vessel Density in Primary Angle Closure Glaucoma: A Quantitative Study Using Optical Coherence Tomography Angiography.

TL;DR: Retinal vessel density was significantly reduced in PACG eyes, and the magnitude of this difference varied between the fundus areas, and was greater in the peripapillary area.
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Doppler optical coherence tomography imaging of local fluid flow and shear stress within microporous scaffolds

TL;DR: Doppler optical coherence tomography is explored as a potential imaging tool for in situ monitoring of local fluid flow profiles inside porous chitosan scaffolds and shows that the scaffold's porosity and interconnectivity can be quantified by combining analyses of the 3D structural and flow images obtained from DOCT.
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Highly sensitive imaging of renal microcirculation in vivo using ultrahigh sensitive optical microangiography.

TL;DR: The capability of UHS-OMAG to provide quantitative analysis about velocity changes in a single capillary vessel (down to tens of microns per second) in response to the ischemic event is attempted.
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In vivo optical imaging of revascularization after brain trauma in mice.

TL;DR: Optical micro-angiography (OMAG) demonstrated that OMAG can differentiate revascularization progression between traumatized mice with and without soluble epoxide hydrolase (sEH) gene deletion, and showed that the sEH knockout effect may be involved in enhancingRevascularization.