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Yin Tintut
Researcher at University of California, Los Angeles
Publications - 124
Citations - 10109
Yin Tintut is an academic researcher from University of California, Los Angeles. The author has contributed to research in topics: Calcification & Osteoblast. The author has an hindex of 43, co-authored 119 publications receiving 9348 citations. Previous affiliations of Yin Tintut include University of California & United States Department of Veterans Affairs.
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Journal ArticleDOI
Vascular Calcification Pathobiology of a Multifaceted Disease
Linda L. Demer,Yin Tintut +1 more
TL;DR: Clinically, vascular calcification is now accepted as a valuable predictor of coronary heart disease, and, conversely, many treatments for cardiovascular disease such as statins, antioxidants, hormone replacement therapy, ACE inhibitors, fish oils, and calcium channel blockers may affect bone health.
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Vascular Calcification. Mechanisms and Clinical Ramifications
TL;DR: Evidence indicates that proteins controlling bone mineralization are also involved in the regulation of vascular calcification, a complex, regulated process of biomineralization resembling osteogenesis, which is widely used as a clinical indicator of atherosclerosis.
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Lipid Oxidation Products Have Opposite Effects on Calcifying Vascular Cell and Bone Cell Differentiation A Possible Explanation for the Paradox of Arterial Calcification in Osteoporotic Patients
Farhad Parhami,Andrew D. Morrow,Jennifer P. Balucan,Norbert Leitinger,Andrew D. Watson,Yin Tintut,Judith A. Berliner,Linda L. Demer +7 more
TL;DR: Results suggest that specific oxidized lipids may be the common factors underlying the pathogenesis of both atherosclerotic calcification and osteoporosis.
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Tumor Necrosis Factor-α Promotes In Vitro Calcification of Vascular Cells via the cAMP Pathway
TL;DR: It is suggested that TNF-&agr; enhances in vitro vascular calcification by promoting osteoblastic differentiation of vascular cells through the cAMP pathway.
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Regulatory mechanisms in vascular calcification
TL;DR: The capacity of the vasculature to produce mineral in culture and to produce de novo, vascularized, trabecular bone and cartilage tissue, even in patients with osteoporosis, should intrigue investigators in tissue engineering and regenerative biology.