Z
Zhuo Xi
Researcher at China Medical University (PRC)
Publications - 19
Citations - 1116
Zhuo Xi is an academic researcher from China Medical University (PRC). The author has contributed to research in topics: Glioma & Gene knockdown. The author has an hindex of 13, co-authored 18 publications receiving 901 citations.
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Journal ArticleDOI
Knockdown of long non-coding RNA XIST exerts tumor-suppressive functions in human glioblastoma stem cells by up-regulating miR-152
Yilong Yao,Jun Ma,Yixue Xue,Ping Wang,Zhen Li,Jing Liu,Liangyu Chen,Zhuo Xi,Hao Teng,Zhenhua Wang,Zhiqing Li,Yunhui Liu +11 more
TL;DR: It is proved that XIST expression was up-regulated in glioma tissues and GSCs and miR-152 mediated the tumor-suppressive effects that knockdown of XIST exerted.
Journal ArticleDOI
TTBK2 circular RNA promotes glioma malignancy by regulating miR-217/HNF1β/Derlin-1 pathway.
TL;DR: It was found that circ-TTBK2 was upregulated in glioma tissues and cell lines, while linear TTBK 2 was not dysregulated inglioma tissue and cells, and circ- TTBk2 acted as miR-217 sponge in a sequence-specific manner.
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Long non-coding RNA taurine upregulated 1 enhances tumor-induced angiogenesis through inhibiting microRNA-299 in human glioblastoma.
TL;DR: It is demonstrated that TUG1 enhances tumor-induced angiogenesis and VEGF expression through inhibiting miR-299 and could provide a novel therapeutic target for glioblastoma treatment.
Journal ArticleDOI
Role of HCP5-miR-139-RUNX1 Feedback Loop in Regulating Malignant Behavior of Glioma Cells.
TL;DR: It is demonstrated that the HCP5-microRNA-139- Runt-related transcription factor 1 feedback loop plays a pivotal role in regulating the malignant behavior of glioma cells, which may provide a potential therapeutic strategy for treating gliomas.
Journal ArticleDOI
Knockdown of long non-coding RNA MALAT1 increases the blood–tumor barrier permeability by up-regulating miR-140
Jun Ma,Ping Wang,Yilong Yao,Yunhui Liu,Zhen Li,Xiaobai Liu,Zhiqing Li,Xihe Zhao,Zhuo Xi,Hao Teng,Jing Liu,Yixue Xue +11 more
TL;DR: The fact that knockdown of MALAT1 resulted in the increased permeability of BTB, which might contribute to establishing potential therapeutic strategies for human gliomas, is demonstrated.