Z
Zsolt Szabó
Researcher at Hungarian Academy of Sciences
Publications - 8
Citations - 156
Zsolt Szabó is an academic researcher from Hungarian Academy of Sciences. The author has contributed to research in topics: Epilepsy & Premovement neuronal activity. The author has an hindex of 6, co-authored 8 publications receiving 105 citations.
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Journal ArticleDOI
Extensive astrocyte synchronization advances neuronal coupling in slow wave activity in vivo
Zsolt Szabó,László Héja,Gergely Szalay,Orsolya Kékesi,András Füredi,Kornélia Szebényi,Árpád Dobolyi,Tamás I. Orbán,Orsolya Kolacsek,Tamás Tompa,Zsombor Miskolczy,László Biczók,Balázs Rózsa,Balázs Sarkadi,Julianna Kardos +14 more
TL;DR: It is demonstrated, for the first time, that the astrocyte network display synchronized recurrent activity in vivo coupled to UP states measured by field recording and neuronal calcium imaging, and conclusively suggest a causal role of theAstrocytic syncytium in SWA generation.
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Recurrent seizure-like events are associated with coupled astroglial synchronization
TL;DR: It is proposed that local, unsynchronized astrocytic Ca2+ transients inhibit, while long-range, synchronized Ca2- signaling contributes to the propagation of recurrent seizure-like events.
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Connexin 43 Differentially Regulates Epileptiform Activity in Models of Convulsive and Non-convulsive Epilepsies.
TL;DR: It is concluded that astrocytic gap junctions are key players in the formation of epileptiform activity and a scheme for the different mode of action in the convulsive and non-convulsive epilepsy types is provided.
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Framing Neuro-Glia Coupling in Antiepileptic Drug Design
TL;DR: From their first detection to their current understanding, various targets that shape both short- and long-range neuro-astroglia coupling are identified, as these are manifest in epilepsy phenomena and in the associated research promotions of AED.
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Feedback adaptation of synaptic excitability via Glu:Na+ symport driven astrocytic GABA and Gln release.
TL;DR: The resulting GABA and Gln release by reverse transport through the respective GAT-3 and SNAT3 transporters help to re-establish the physiological Na+ homeostasis without ATP dissipation and consequently leads to enhanced tonic inhibition and replenishment of axonal glutamate pool.