Z
Zsuzsanna Fabry
Researcher at University of Wisconsin-Madison
Publications - 66
Citations - 2764
Zsuzsanna Fabry is an academic researcher from University of Wisconsin-Madison. The author has contributed to research in topics: Immune system & T cell. The author has an hindex of 27, co-authored 62 publications receiving 2362 citations. Previous affiliations of Zsuzsanna Fabry include University of Wisconsin Hospital and Clinics.
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Journal ArticleDOI
Probiotic helminth administration in relapsing–remitting multiple sclerosis: a phase 1 study:
John O. Fleming,Isaak A,Lee Je,Christopher Luzzio,Carrithers,Thomas D. Cook,Aaron S. Field,Boland J,Zsuzsanna Fabry +8 more
TL;DR: TSO was well tolerated in the first human study of this novel probiotic in RRMS, and favorable trends were observed in exploratory MRI and immunological assessments.
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Initiation of Immune Responses in Brain Is Promoted by Local Dendritic Cells
TL;DR: It is shown that cells expressing CD11c, CD205, and MHC class II molecules and containing fluorescently labeled, processed Ag accumulate at the site of intracerebral Ag injection, and that brain-emigrant DCs are sufficient to support activated T cells to home to the tissue of DC origination.
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Interleukin-6 promotes post-traumatic healing in the central nervous system.
Karin R. Swartz,Frances Liu,Diane Sewell,Terri L. Schochet,Iain L. Campbell,Matyas Sandor,Zsuzsanna Fabry +6 more
TL;DR: The accelerated tissue repair in GFAP-IL-6 transgenic animals is primarily due to extensive re-vascularization as detected by endothelial cell markers, which suggests an important role of IL-6 in tissue repair processes following traumatic injury in the CNS.
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Brain endothelial cell production of a neuroprotective cytokine, interleukin-6, in response to noxious stimuli
TL;DR: A pathway whereby immune signals may be communicated to the CNS is indicated and one way that the BBB may protect neuronal survival under challenge conditions is revealed.
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Dendritic Cell Transmigration through Brain Microvessel Endothelium Is Regulated by MIP-1α Chemokine and Matrix Metalloproteinases
TL;DR: It is reported that MIP-1α increases the transmigration of bone marrow-derived, GFP-labeled DCs across brain microvessel endothelial cell monolayers and shows that DCs produce matrix metalloproteinases (MMP) -2 and -9 and GM6001, an MMP inhibitor, decreases both baseline and MIP -1α-induced DC transmigration.