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Burden of Disease from Environmental Noise: Quantification of Healthy Life Years Lost in Europe

TLDR
Policy-makers and their advisers are provided with technical support in their quantitative risk assessment of environmental noise and can use the procedure for estimating burdens presented here to prioritize and plan environmental and public health policies.
Abstract
The health impacts of environmental noise are a growing concern. At least one million healthy life years are lost every year from traffic-related noise in the western part of Europe. This publication summarises the evidence on the relationship between environmental noise and health effects, including cardiovascular disease, cognitive impairment, sleep disturbance, tinnitus, and annoyance. For each one, the environmental burden of disease methodology, based on exposure-response relationship, exposure distribution, background prevalence of disease and disability weights of the outcome, is applied to calculate the burden of disease in terms of disability-adjusted life-years. Data are still lacking for the rest of the WHO European Region. This publication provides policy-makers and their advisers with technical support in their quantitative risk assessment of environmental noise. International, national and local authorities can use the procedure for estimating burdens presented here to prioritize and plan environmental and public health policies.

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Citations
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Auditory and non-auditory eff ects of noise on health

TL;DR: In this paper, the authors stress the importance of adequate noise prevention and mitigation strategies for public health and stress that noise exposure leads to annoyance, disturbs sleep and causes daytime sleepiness, aff ects patient outcomes and staff performance in hospitals, increases the occurrence of hypertension and cardiovascular disease, and impairs cognitive performance in schoolchildren.
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A synthesis of two decades of research documenting the effects of noise on wildlife

TL;DR: A systematic and standardised review of the scientific literature published from 1990 to 2013 on the effects of anthropogenic noise on wildlife, including both terrestrial and aquatic studies shows that terrestrial wildlife responses begin at noise levels of approximately 40’dBA, and 20% of papers documented impacts below 50 dBA.
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Cardiovascular effects of environmental noise exposure

TL;DR: Evidence from epidemiologic studies demonstrates that environmental noise is associated with an increased incidence of arterial hypertension, myocardial infarction, and stroke, and the importance of noise mitigation strategies for public health is stressed.
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Urban natural environments as nature-based solutions for improved public health – A systematic review of reviews

TL;DR: The results show that there is strong evidence for improved affect as well as on heat reduction from urban natural environments, and guidelines on how public health and well-being could be integrated into implementation of NBS for resilient and liveable urban landscapes and health in a changing climate are provided.
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WHO Environmental Noise Guidelines for the European Region: A Systematic Review on Environmental Noise and Effects on Sleep.

TL;DR: Based on the available evidence, transportation noise affects objectively measured sleep physiology and subjectively assessed sleep disturbance in adults and for children’s sleep.
References
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Lung Cancer, Cardiopulmonary Mortality, and Long-term Exposure to Fine Particulate Air Pollution

TL;DR: Fine particulate and sulfur oxide--related pollution were associated with all-cause, lung cancer, and cardiopulmonary mortality and long-term exposure to combustion-related fine particulate air pollution is an important environmental risk factor for cardiopULmonary and lung cancer mortality.
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TL;DR: The evidence for adverse effects on health of selected air pollutants is discussed, and it is unclear whether a threshold concentration exists for particulate matter and ozone below which no effect on health is likely.
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Stress, adaptation, and disease. Allostasis and allostatic load.

TL;DR: The relationship of allostatic load to genetic and developmental predispositions to disease is considered and examples will be given from research pertaining to autonomic, CNS, neuroendocrine, and immune system activity.
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