Journal ArticleDOI
Hyperosmolar hyperglycemic nonketotic diabetic coma.
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This article is published in Medical Clinics of North America.The article was published on 1970-05-01. It has received 119 citations till now. The article focuses on the topics: Diabetic coma.read more
Citations
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Journal ArticleDOI
The Hypertonic State
Peter U. Feig,Donna K. Mccurdy +1 more
TL;DR: A network of regulatory systems that stabilize body-fluid volume, temperature and composition and insulates cells from an ambient hostile world and provides stability in the face of environmental change are proposed.
Journal ArticleDOI
Clinical and metabolic characteristics of hyperosmolar nonketotic coma.
TL;DR: It is suggested that dehydration and hyperosmolarity may play significant roles in the etiology of HNC, and that therapy should, therefore, be directed at restoration of normal osmolarity and correction of water deficits with 0.45 per cent saline and moderate amounts of insulin.
Journal ArticleDOI
Nutritional support of the hospitalized patient. The team concept.
TL;DR: The study concludes that a protocol strictly adhered to by knowledgeable persons is necessary for the proper administration of TPN in the hospitalized patient.
Journal ArticleDOI
Hypouricemia in the Syndrome of Inappropriate Secretion of Antidiuretic Hormone
TL;DR: A retrospective study of serum levels of uric acid in hyponatremic patients was performed and indicates not only that hypouricemia is the rule in patients with inappropriate secretion of antidiuretic hormone but also that the serum concentrations of urate can be affected.
Journal ArticleDOI
Hyponatremia and hypernatremia.
Linda F. Fried,Paul M. Palevsky +1 more
TL;DR: The primary treatment of hypernatremia is water replacement-repleting water deficits and replacing ongoing losses, and additional treatment should be directed at eliminating excess water losses.
References
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Journal ArticleDOI
Effect of Very Small Concentrations of Insulin on Forearm Metabolism. Persistence of Its Action on Potassium and Free Fatty Acids without Its Effect on Glucose
Journal Article
Pathogenesis of lesions in the nervous system in hypernatremic states. II. Experimental studies of gross anatomic changes and alterations of chemical composition of the tissues.
TL;DR: A study of the inorganic chemical composition of the tissues revealed a difference in the pattern of osmotic adjustment between cells of brain and of muscle that may in part account for the marked manifestations related to the nervous system seen in the animals and, by analogy, in patients.
Journal ArticleDOI
Factors that limit brain volume changes in response to acute and sustained hyper- and hyponatremia.
TL;DR: The brain protects itself from acute volume changes in response to change in Na(E) by the freedom for Na and Cl to move from the Cl space, by V(i) not changing acutely to the degree predicted from osmotic properties of cells in general, and by significant quantities of Na and K in brain are osmotically inactive.
Journal ArticleDOI
Focal seizures as a manifestation of hyperglycemia without ketoacidosis. a report of seven cases with review of the literature.
TL;DR: Nineteen cases of nonketoacidotic hyperglycemia reported since 1956 and one additional case which, in the authors' opinion, represents an example of this entity are summarized in table 1.
Journal ArticleDOI
Neurological dysfunction associated with nonketotic hyperglycemia.
TL;DR: The metabolic abnormalities that define the syn drome have been delineated and the necessity for its early recognition and prompt treatment with large quantities of hypotonic fluids and insulin is well established.
Related Papers (5)
Nonketotic hyperosmolar coma with hyperglycemia: clinical features, pathophysiology, renal function, acid-base balance, plasma-cerebrospinal fluid equilibria and the effects of therapy in 37 cases.
Allen I. Arieff,Hugh J. Carroll +1 more