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Open AccessJournal Article

Physiology of the heart

Ulrich J
- 01 Jul 1950 - 
- Vol. 39, pp 408-417
TLDR
In the single authored, expert voice that is this book's unique strength, Arnold Katz provides a comprehensive overview of the physiological and biophysical basis of cardiac function, beginning with structure and proceeding to biochemistry, biophysics, and pathophysiology in arrhythmias, ischemia, and heart failure.
Abstract
In the single authored, expert voice that is this book's unique strength, Arnold Katz provides a comprehensive overview of the physiological and biophysical basis of cardiac function, beginning with structure and proceeding to biochemistry, biophysics, and pathophysiology in arrhythmias, ischemia, and heart failure. Emphasis is on the interrelationships of basic processes among the cell, cardiac muscle function, and the biophysics of contractile and electrical behavior. This edition includes new material on cell signaling and molecular biology.

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Citations
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Cardiac Metabolism and its Interactions With Contraction, Growth, and Survival of Cardiomyocytes

TL;DR: An overview of the cardiac metabolic network is provided and alterations observed in cardiac pathologies as well as strategies used as metabolic therapies in heart failure are highlighted.
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Energy metabolism in heart failure

TL;DR: This review will focus on energy metabolism alterations in long‐term chronic heart failure with only a few references to compensated hypertrophy when necessary and will briefly describe the energy metabolism of normal heart and skeletal muscles and their alterations in chronicheart failure.
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Regulation of cardiac ion channels by catecholamines, acetylcholine and second messenger systems.

TL;DR: The results confirmed the importance of knowing the carrier and removal status of canine coronavirus, as a source of infection for other animals, not necessarily belonging to the same breeds.
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T-Tubule Function in Mammalian Cardiac Myocytes

TL;DR: The t‐tubules are an important determinant of cardiac cell function, especially as the main site of excitation‐contraction coupling, ensuring spatially and temporally synchronous Ca2+ release throughout the cell.
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Time to move beyond a brainless exercise physiology: the evidence for complex regulation of human exercise performance.

TL;DR: It is argued that the central governor model (CGM) is better able to explain at least 6 common phenomena, including differential pacing strategies for different exercise durations, the end spurt, and the presence of fatigue even though homeostasis is maintained.
References
More filters
Journal ArticleDOI

Cardiac Metabolism and its Interactions With Contraction, Growth, and Survival of Cardiomyocytes

TL;DR: An overview of the cardiac metabolic network is provided and alterations observed in cardiac pathologies as well as strategies used as metabolic therapies in heart failure are highlighted.
Journal ArticleDOI

Energy metabolism in heart failure

TL;DR: This review will focus on energy metabolism alterations in long‐term chronic heart failure with only a few references to compensated hypertrophy when necessary and will briefly describe the energy metabolism of normal heart and skeletal muscles and their alterations in chronicheart failure.
Journal ArticleDOI

Regulation of cardiac ion channels by catecholamines, acetylcholine and second messenger systems.

TL;DR: The results confirmed the importance of knowing the carrier and removal status of canine coronavirus, as a source of infection for other animals, not necessarily belonging to the same breeds.
Journal ArticleDOI

T-Tubule Function in Mammalian Cardiac Myocytes

TL;DR: The t‐tubules are an important determinant of cardiac cell function, especially as the main site of excitation‐contraction coupling, ensuring spatially and temporally synchronous Ca2+ release throughout the cell.
Journal ArticleDOI

Time to move beyond a brainless exercise physiology: the evidence for complex regulation of human exercise performance.

TL;DR: It is argued that the central governor model (CGM) is better able to explain at least 6 common phenomena, including differential pacing strategies for different exercise durations, the end spurt, and the presence of fatigue even though homeostasis is maintained.