What are the pharmacological properties of polygonatum orientale?
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Polygonatum orientale, a traditional Chinese medicinal plant, exhibits various pharmacological properties. It has been found to have beneficial effects in treating myocardial ischemia (MI) by activating the MAPK/ERK signaling pathway, reducing ST segment elevation, heart tissue injury, and inflammatory markers. Additionally, Polygonatum orientale contains bioactive compounds like phenolic and flavonoids, which contribute to its antioxidant and antibacterial activities, making it a potential option for treating Staphylococcus aureus infections. Furthermore, the plant's anti-inflammatory and analgesic effects have been attributed to flavonoids like orientin, isoorientin, vitexin, luteolin, and quercetin, which inhibit inflammatory responses and pain sensations. Overall, Polygonatum orientale demonstrates a wide range of pharmacological properties, including anti-inflammatory, antioxidant, antibacterial, and cardioprotective effects.
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14 Citations | Polygonatum orientale contains steroidal saponins, polysaccharides, flavonoids, and alkaloids, exhibiting anti-aging, anti-tumor, immunomodulatory, antibacterial, antiviral, hypoglycemic, and blood lipid effects, as highlighted in the research. |
34 Citations | Polygonum orientale exhibits anti-inflammatory and analgesic effects due to flavonoids like orientin and quercetin, reducing TNF-α, IL-1β, IL-6, and PGE2 concentrations in serum. |
| Polygonatum orientale contains high levels of phenolic and flavonoid compounds, exhibiting antioxidant and antibacterial properties, making it a potential option for pharmacological studies in treating Staphylococcus aureus infections. | |
| Polygonum orientale L. exhibits antimyocardial ischemia (MI) properties. Its main compounds include isoorientin, orientin, vitexin, quercitrin, astragalin, and protocatechuic acid, showing potential pharmacological activities. | |
| Polygonum orientale L. exhibits pharmacological properties in alleviating myocardial ischemia-induced injury by activating the MAPK/ERK signaling pathway, reducing apoptosis, inflammation, and improving cardiac function. |
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