Showing papers on "Hypoventilation published in 1970"

Effect of intravenous alprenolol on ventilatory function and blood gas values in established obstructive airways disease

Abstract: Intravenous administration of the beta-adrenergic blocking drug, alprenolol, to a group of patients with severe obstructive disease of the airways caused no deterioration of symptoms and a clinically insignificant fall in FEV1 in the majority. A minor reduction in PaO2 occurred which suggests that alprenolol may cause local hypoventilation and/or pulmonary arteriovenous shunting. The presence of obstructive disease of the airways does not seem to pose an absolute contraindication to the use of alprenolol, although caution must be exercised in its use in such patients.

Oxygen therapy in acute respiratory failure.

Abstract: SIR: I would like t.o comment on the letter from John Read in the Journal on November 28, 1970, concerning a paper by Vandenberg et alii published in the Journal on November 7, 1970. The letter raised several points of considerable theoretical interest. The authors of the article were not unaware of the points raised; however, consideration of them was not the purpose of the paper, nor do we believe that the facts available permitted discussion of them.

Postoperative hypoventilation and hypoxemia in man after hyperventilation

Abstract: In 13 patients, free of cardiopulmonary disease, who had been subjected to hyperventilation for an average of two and three-quarters hours during anesthesia and operation, spontaneous breathing began when arterial carbon dioxide tension rose from 18 to 38 mm of mercury. Thereafter the patients were allowed to breathe air. Tidal volume and minute volume increased progressively throughout the first postoperative hour while arterial carbon dioxide tension varied less than 2 mm of mercury. Twenty minutes after the onset of spontaneous breathing, arterial oxygen tension reached an average low value of 72 mm of mercury and then progressively increased toward normal, averaging 83 mm at 30 minutes and 88 mm at 60 minutes. Thus, in man the hypoventilation that follows a period of hyperventilation can result in a definite lowering of arterial oxygen tension if air is breathed after operation. Yet this hypoventilation was associated with a normal arterial carbon dioxide tension. Carbon dioxide tension is no...

CASE HISTORY: Hypokalemia as a Cause of Hypoventilation During Anesthesia for Burns

Abstract: 64-YEAR-OLD white housewife suffered It was estimated that the burns, about half A extensive bums to the palms of both of which were second degree and half third hands, her right side, both thighs posteriorly, degree, covered approximately 25 percent buttocks, shoulders, and the lower third of of the total body surface. Treatment conthe back on November 21, 1964. At her sisted of Hubbard tank* therapy daily for community hospital she received parenteral the first 3 weeks of hospitalization. Electrofluids, intravenous penicillin prophylacticlytes were kept within normal limits by ally, and narcotic drugs to control the pain. appropriate parenteral fluid administration when nausea developed 4 hours after hosand diet. On December 24, the wounds first pitaluation, Compazine@ was administered appeared to be infected and, as Pseudomofor its control. The only local medication nas was cultured, Coly-mycina and proto the bums was 1 percent Furacin.\" caine penicillin therapy was started.