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Alfred Sommer
Researcher at Johns Hopkins University
Publications - 366
Citations - 32936
Alfred Sommer is an academic researcher from Johns Hopkins University. The author has contributed to research in topics: Xerophthalmia & Vitamin A deficiency. The author has an hindex of 86, co-authored 364 publications receiving 31556 citations. Previous affiliations of Alfred Sommer include Centers for Disease Control and Prevention & Johns Hopkins University School of Medicine.
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Journal ArticleDOI
Effects of vitamin A deficiency on the ocular surface
TL;DR: Vitamin A deficiency remains an important cause of ocular morbidity among patients with chronic liver disease and lipid malabsorption, and is a major cause of blindness in developing countries.
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Impression cytology: a practical index of vitamin A status.
TL;DR: Impression cytology was performed on 148 Indonesian preschool children of whom half had mild xerophthalmia and half were age-matched control subjects and 12 of 26 clinically normal children with serum vitamin A levels less than 20 micrograms/dL had abnormal impression cytology.
Journal ArticleDOI
Ocular BB injuries.
TL;DR: Data from the United States Consumer Product Safety Commission indicate that air-powered guns were responsible for 22,800 injuries treated in emergency rooms during 1981, of which 1255 were eye injuries.
Journal ArticleDOI
Interobserver and intraobserver variability in the detection of glaucomatous progression of the optic disc.
Anne L. Coleman,Alfred Sommer,Cheryl Enger,Harry L.S. Knopf,Robert L. Stamper,Donald S. Minckler +5 more
TL;DR: Baseline stereoscopic photographs of the optic nerve head did not substantially improve recognition of progressive glaucomatous optic nerve damage when compared with the use of baseline drawings of the optics nerve head made from photographs in subjects who developed visual loss in the interim.
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Screening for Glaucomatous Visual Field Loss: The Effect of Patient Reliability
Joanne Katz,Alfred Sommer +1 more
TL;DR: Glaucoma patients with poor fixation had less depressed fields and fewer localized defects than those with good fixation, and high false-positive rates were associated with less-depressed visual fields among glaucomA patients and normal subjects.