A
Aminah Jatoi
Researcher at Mayo Clinic
Publications - 440
Citations - 15796
Aminah Jatoi is an academic researcher from Mayo Clinic. The author has contributed to research in topics: Cancer & Lung cancer. The author has an hindex of 46, co-authored 404 publications receiving 12708 citations. Previous affiliations of Aminah Jatoi include University of Texas Health Science Center at San Antonio & University of North Carolina at Chapel Hill.
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Journal ArticleDOI
Role of dietary carbohydrates on risk of lung cancer.
Jun Tao,Aminah Jatoi,Jeffrey Crawford,Wendy W. T. Lam,James Chung-Man Ho,Xiaofei Wang,Herbert Pang,Herbert Pang +7 more
TL;DR: In this paper, the role of dietary carbohydrates on lung cancer risk was comprehensively evaluated, and a multivariable logistic model adjusted for confounders was used to estimate odds ratios (ORs) and 95 % confidence intervals (CIs) for dietary carbohydrates, fiber, whole grains, glycemic index (GI) and glycemic load (GL) for lung cancer.
Journal Article
Palliating hiccups in cancer patients: moving beyond recommendations from Leonard the lion.
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Lymphedema, musculoskeletal events and arm function in older patients receiving adjuvant chemotherapy for breast cancer (Alliance A171302)
Judith O. Hopkins,Jake Allred,Arti Hurria,Aminah Jatoi,Jacqueline M. Lafky,Harvey J. Cohen,Clifford A. Hudis,Eric P. Winer,J. Mandelblatt,Ann H. Partridge,Lisa A. Carey,Hyman B. Muss +11 more
TL;DR: Potentially debilitating MEs occur in three-fourths of elderly women undergoing standard therapy for breast cancer, and emphasis should be placed on prevention, identification, and treatment of these MEs to improve QOL.
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Is ATP (Adenosine 5′-Triphosphate), like STP®, a Performance-Enhancing Additive for the Tanks of Cancer Patients?
TL;DR: A randomized, controlled trial studying adenosine 5’-triphosphate (ATP) infusions as therapy for cancer anorexia/cachexia in patients with non-small-cell lung cancer is described, seeking the effect of ATP infusions on body weight change, muscle strength, serum albumin concentration, and quality-oflife scores.
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Increased incidence of endometrioid tumors caused by aberrations in E-cadherin promoter of mismatch repair-deficient mice.
TL;DR: It is shown that combined disruption of E-cadherin and DNA MMR pathways increases incidence of endometrioid tumors in mice and Msh2(-/-)/Cdh1(+/-) mice provide a good model of gynecological tumorigenesis and may be useful for testing molecular target-specific therapies.