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Amit Kumar

Researcher at Shenzhen University

Publications -  319
Citations -  17696

Amit Kumar is an academic researcher from Shenzhen University. The author has contributed to research in topics: Photocatalysis & Adsorption. The author has an hindex of 63, co-authored 256 publications receiving 12509 citations. Previous affiliations of Amit Kumar include Sun Microsystems & Glocal University.

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Oxidative Stress, Prooxidants, and Antioxidants: The Interplay

TL;DR: The importance of oxidative stress in the body growth and development as well as proteomic and genomic evidences of its relationship with disease development, incidence of malignancies and autoimmune disorders, increased susceptibility to bacterial, viral, and parasitic diseases are discussed.
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BIM Mediates EGFR Tyrosine Kinase Inhibitor-Induced Apoptosis in Lung Cancers with Oncogenic EGFR Mutations

TL;DR: Evidence is provided that BIM is involved in TKI- induced apoptosis in sensitive EGFR-mutant cells and that both attenuation of the up-regulation of BIM and resistance to gefitinib-induced apoptosis are seen in models that contain the common EGFR T790M and the novel L747S secondary resistance mutations.
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Novel development of nanoparticles to bimetallic nanoparticles and their composites: A review

TL;DR: Bimetallic nanoparticles (BNPs) are formed by the combination of two different metals and have attracted huge attention in both technological and scientific view because BNPs shows better properties as mentioned in this paper.
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A review of the mechanical and thermal properties of graphene and its hybrid polymer nanocomposites for structural applications

TL;DR: In this paper, a review of the recent progress in describing the intricacies of mechanical and thermal properties of all types of graphene and modified graphene-based polymer nanocomposites has been comprehensively examined.
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Structure and clinical relevance of the epidermal growth factor receptor in human cancer

TL;DR: An atomic-level mechanism is suggested for the poor efficacy of lapatinib against tumors with activating EGFR kinase domain point mutations compared with the efficacy of gefitinib and erlotinib, and how structural insights help the understanding of acquired resistance to these agents.