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Ana Luiza Maia
Researcher at Universidade Federal do Rio Grande do Sul
Publications - 175
Citations - 6890
Ana Luiza Maia is an academic researcher from Universidade Federal do Rio Grande do Sul. The author has contributed to research in topics: Thyroid & Deiodinase. The author has an hindex of 36, co-authored 160 publications receiving 5736 citations. Previous affiliations of Ana Luiza Maia include Boston Medical Center & Yahoo!.
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Journal ArticleDOI
2016 American Thyroid Association Guidelines for Diagnosis and Management of Hyperthyroidism and Other Causes of Thyrotoxicosis
Douglas S. Ross,Henry B. Burch,David S. Cooper,M. Carol Greenlee,Peter Laurberg,Ana Luiza Maia,Scott A. Rivkees,Mary H. Samuels,Julie Ann Sosa,Marius N. Stan,Martin A. Walter +10 more
TL;DR: One hundred twenty-four evidence-based recommendations were developed to aid in the care of patients with thyrotoxicosis and to share what the task force believes is current, rational, and optimal medical practice.
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Type 2 iodothyronine deiodinase is the major source of plasma T3 in euthyroid humans
TL;DR: Impairment of D2-generated T3 is the major cause of the reduced T3 production in the euthyroid sick syndrome, and it is suggested that generation of nuclear T 3 is an intrinsic property of the D2 protein.
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The role of thyroid hormone in testicular development and function.
TL;DR: The current review aims at presenting an updated picture of the recent advances made regarding the role of thyroid hormones in male gonadal function.
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The type 2 deiodinase A/G (Thr92Ala) polymorphism is associated with decreased enzyme velocity and increased insulin resistance in patients with type 2 diabetes mellitus.
Luis Henrique Santos Canani,Clarissa Capp,José Miguel Dora,Erika L. Souza Meyer,Márcia dos Santos Wagner,John W. Harney,P. Reed Larsen,Jorge Luiz Gross,Antonio C. Bianco,Ana Luiza Maia +9 more
TL;DR: The A/G polymorphism is associated with greater insulin resistance in type 2 diabetes mellitus patients and with lower D2 velocity in tissue samples, suggesting that the D2-generated T(3) in skeletal muscle plays a role in insulin resistance.
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Type 3 lodothyronine deiodinase: cloning, in vitro expression, and functional analysis of the placental selenoenzyme.
Domenico Salvatore,S C Low,M J Berry,Ana Luiza Maia,John W. Harney,Walburga Croteau,D L St Germain,P R Larsen +7 more
TL;DR: The conservation of this enzyme from Xenopus laevis tadpoles to humans implies an essential role for regulation of thyroid hormone inactivation during embryological development.