A
Anders F. Andersson
Researcher at Royal Institute of Technology
Publications - 188
Citations - 20568
Anders F. Andersson is an academic researcher from Royal Institute of Technology. The author has contributed to research in topics: Homocysteine & Metagenomics. The author has an hindex of 60, co-authored 176 publications receiving 17400 citations. Previous affiliations of Anders F. Andersson include Karolinska Institutet & Uppsala University.
Papers
More filters
Journal ArticleDOI
The impact of Crohn's disease genes on healthy human gut microbiota: a pilot study
Christopher Quince,Elin E Lundin,Anna Andreasson,Anna Andreasson,Dario Greco,Joseph Rafter,Nicholas J. Talley,Lars Agréus,Anders F. Andersson,Lars Engstrand,Mauro D'Amato +10 more
TL;DR: The impact of 30 unequivocal CD-risk loci, each tagged by a single nucleotide polymorphism (SNP), on the mucosa-associated bacteria in healthy individuals was studied.
Journal ArticleDOI
Prevalent reliance of bacterioplankton on exogenous vitamin B1 and precursor availability
Ryan W. Paerl,John Sundh,Demeng Tan,Sine Lo Svenningsen,Samuel Hylander,Jarone Pinhassi,Anders F. Andersson,Lasse Riemann +7 more
TL;DR: It is shown that B1 auxotrophy, the need for exogenous B1 or precursors for survival, is widespread among wild bacterioplankton, and a hitherto overlooked influence of B1/precursor availability on aquatic biochemical cycling is suggested.
Journal ArticleDOI
Ecosystem-wide metagenomic binning enables prediction of ecological niches from genomes.
Johannes Alneberg,Christin Bennke,Sara Beier,Sara Beier,Carina Bunse,Carina Bunse,Carina Bunse,Christopher Quince,Karolina Ininbergs,Karolina Ininbergs,Lasse Riemann,Martin Ekman,Klaus Jürgens,Matthias Labrenz,Jarone Pinhassi,Anders F. Andersson +15 more
TL;DR: This study conducts metagenomics binning of water samples collected over major environmental gradients in the Baltic Sea and uses machine-learning to predict the placement of genome clusters along niche gradients based on the content of functional genes.
Journal ArticleDOI
The influence of hyperglycaemia, hyperinsulinaemia and genetic background on the fate of intrasplenically implanted mouse islets
TL;DR: Neither hyperglycaemia nor hyperinsulinaemia per se are primarily responsible for the growth of islets in obese-hyperglycaemic mice, and the present data suggest that the genetic background is very important for morphological and functional responses of the islets to a prolonged period of hyper glycaemic stress.
Journal ArticleDOI
Hypomethylation as a cause of homocysteine-induced cell damage in human cell lines.
TL;DR: A combination of two potentially cell-damaging mechanisms (formation of oxygen radicals and hypomethylation) aggravated the retardation of cell growth compared to only one of these mechanisms being present, suggesting that several mechanisms of homocysteine-induced cell damage contribute to the increased rate of the atherogenic process observed in hyperhomocysteinemia.